Free Access
Med Sci (Paris)
Volume 25, Number 10, Octobre 2009
Page(s) 773 - 774
Section Nouvelles
Published online 15 October 2009
  1. Herczenik E, Gebbink MF. Molecular and cellular aspects of protein misfolding and disease. FASEB J 2008; 22 : 2115–33. [Google Scholar]
  2. Ren PH, Lauckner JE, Kachirskaia I, et al. Cytoplasmic penetration and persistent infection of mammalian cells by polyglutamine aggregates. Nat Cell Biol 2009; 11 : 219–25. [Google Scholar]
  3. Luo S, Mizuta H, Rubinsztein DC. p21-activated kinase 1 promotes soluble mutant huntingtin self-interaction and enhances toxicity. Hum Mol Genet 2008; 17 : 895–905. [Google Scholar]
  4. Zhang H, Li Q, Graham RK, et al. Full length mutant huntingtin is required for altered Ca2+ signaling and apoptosis of striatal neurons in the YAC mouse model of Huntington’s disease. Neurobiol Dis 2008; 31 : 80–8. [Google Scholar]
  5. Lelan F, Damier P. Les neurones dopaminergiques greffés dans la maladie de Parkinson sont-il à leur tour atteints par le processus dégénératif ? Med Sci (Paris) 2009; 25 : 15–6. [Google Scholar]
  6. Li JY, Englund E, Holton JL, et al. Lewy bodies in grafted neurons in subjects with Parkinson’s disease suggest host-to-graft disease propagation. Nat Med 2008; 14 : 501–3. [Google Scholar]

Current usage metrics show cumulative count of Article Views (full-text article views including HTML views, PDF and ePub downloads, according to the available data) and Abstracts Views on Vision4Press platform.

Data correspond to usage on the plateform after 2015. The current usage metrics is available 48-96 hours after online publication and is updated daily on week days.

Initial download of the metrics may take a while.