EDP Sciences logo
Subscriber Authentication Point
Search
Menu
Home All issues Volume 19 / No 12 (Décembre 2003) Med Sci (Paris), 19 12 (2003) 1233-1241 References
Free Access
Issue
Med Sci (Paris)
Volume 19, Number 12, Décembre 2003
Page(s) 1233 - 1241
Section M/S revues
DOI https://doi.org/10.1051/medsci/200319121233
Published online 15 December 2003
  1. Galla JH. IgA nephropathy. Kidney Int 1995; 47 : 377–87. [Google Scholar]
  2. Simon P, Ramee MP, Autuly V, et al. Epidemiology of primary glomerular diseases in a French region. Variations according to period andage. Kidney Int 1994; 46 : 1192–8. [Google Scholar]
  3. Berger J, Hinglais N. Les dépôts intercapillaires d’IgA-IgG. J Urol Néphrol 1968; 74 : 694–5. [Google Scholar]
  4. Aucouturier P, Monteiro RC, Noel LH, Preud’homme JL, Lesavre P. Glomerular and serum immunoglobulin G subclasses in IgA nephropathy. Clin Immunol Immunopathol 1989; 51 : 338–47. [Google Scholar]
  5. Meadow SR, Scott DG. Berger disease : Henoch-Schonlein syndrome without the rash. J Pediatr 1985; 106 : 27–32. [Google Scholar]
  6. Berger J, Yaneva H, Nabarra B, Barbanel C. Recurrence of mesangial deposition of IgA after renal transplantation. Kidney Int 1975; 7 : 232–41. [Google Scholar]
  7. Ponticelli C, Traversi L, Feliciani A, et al. Kidney transplantation in patients with IgA mesangial glomerulonephritis. Kidney Int 2001; 60 : 1948–54. [Google Scholar]
  8. Berger J. Glomérulonéphrites idiopathiques à dépôts mésangiaux d’IgA. In : Hamburger J, Crosnier J, Grünfeld JP, eds. Néphrologie. Paris : Flammarion/Médicine-Sciences, 1979 : 541–8. [Google Scholar]
  9. Valentijn RM, Radl J, Haaijman JJ, et al. Circulating and mesangial secretory component-binding IgA-1 in primary IgA nephropathy. Kidney Int 1984; 26 : 760–6. [Google Scholar]
  10. Monteiro RC, Halbwachs-Mecarelli L, Roque-Barreira MC, et al. Charge and size of mesangial IgA in IgA nephropathy. Kidney Int 1985; 28 : 666–71. [Google Scholar]
  11. Rifai A, Small PA Jr, Teague PO, Ayoub EM. Experimental IgA nephropathy. J Exp Med 1979; 150 : 1161–73. [Google Scholar]
  12. van den Wall Bake AW, Bruijn JA, Accavitti MA, et al. Shared idiotypes in mesangial deposits in IgA nephropathy are not disease-specific. Kidney Int 1993; 44 : 65–74. [Google Scholar]
  13. de Fijter JW, Eijgenraam JW, Braam CA, et al. Deficient IgA1 immune response to nasal cholera toxin subunit B in primary IgA nephropathy. Kidney Int 1996; 50 : 952–61. [Google Scholar]
  14. Barratt J, Bailey EM, Buck KS, et al. Exaggerated systemic antibody response to mucosal Helicobacter pylori infection in IgA nephropathy. Am J Kidney Dis 1999; 33 : 1049–57. [Google Scholar]
  15. Layward L, Allen AC, Harper SJ, Hattersley JM, Feehally J. Increased and prolonged production of specific polymeric IgA after systemic immunization with tetanus toxoid in IgA nephropathy. Clin Exp Immunol 1992; 88 : 394–8. [Google Scholar]
  16. Feehally J. IgA nephropathy : a disorder of IgA production ? Q J Med 1997; 90 : 387–90. [Google Scholar]
  17. Allen AC, Bailey EM, Brenchley PE, et al. Mesangial IgA1 in IgA nephropathy exhibits aberrant O-glycosylation : observations in three patients. Kidney Int 2001; 60 : 969–73. [Google Scholar]
  18. Allen AC, Willis FR, Beattie TJ, Feehally J. Abnormal IgA glycosylation in Henoch-Schonlein purpura restricted to patients with clinical nephritis. Nephrol Dial Transplant 1998; 13 : 930–4. [Google Scholar]
  19. Sano T, Hiki Y, Kokubo T, et al. Enzymatically deglycosylated human IgA1 molecules accumulate and induce inflammatory cell reaction in rat glomeruli. Nephrol Dial Transplant 2002; 17 : 50–6. [Google Scholar]
  20. Greer MR, Barratt J, Harper SJ, Allen AC, Feehally J. The nucleotide sequence of the IgA1 hinge region in IgA nephropathy. Nephrol Dial Transplant 1998; 13 : 1980–3. [Google Scholar]
  21. Hiki Y, Tanaka A, Kokubo T, et al. Analyses of IgA1 hinge glycopeptides in IgA nephropathy by matrix-assisted laser desorption/ionization time-of-flight mass spectrometry. J Am Soc Nephrol 1998; 9 : 577–82. [Google Scholar]
  22. Allen AC, Topham PS, Harper SJ, Feehally J. Leucocyte beta 1,3 galactosyltransferase activity in IgA nephropathy. Nephrol Dial Transplant 1997; 12 : 701–6. [Google Scholar]
  23. Kokubo T, Hiki Y, Iwase H, et al. Protective role of IgA1 glycans against IgA1 self-aggregation and adhesion to extracellular matrix proteins. J Am Soc Nephrol 1998; 9 : 2048–54. [Google Scholar]
  24. Tomana M, Novak J, Julian BA, Matousovic K, Mestecky J. Circulating immune complexes in IgA nephropathy consist of IgA1 with galactose-deficient hinge region and antiglycan antibodies. J Clin Invest 1999; 104 : 73–81. [Google Scholar]
  25. Novak J, Julian BA, Tomana M, Mestecky J. Progress in molecular and genetic studies of IgA nephropathy. J Clin Immunol 2001; 21 : 310–27. [Google Scholar]
  26. Monteiro RC, van de Winkel JGJ. IgA Fc receptors. Annu Rev Immunol 2003; 21 : 177–204. [Google Scholar]
  27. Grossetete B, Launay P, Lehuen A, et al. Down-regulation of Fca receptors on blood cells of IgA nephropathy patients: evidence for a negative regulatory role of serum IgA. Kidney Int 1998; 53 : 1321–35. [Google Scholar]
  28. Launay P, Grossetete B, Arcos-Fajardo M, et al. Fca receptor (CD89) mediates the development of immunoglobulin A (IgA) nephropathy (Berger’s disease). Evidence for pathogenic soluble receptor-IgA complexes in patients and CD89 transgenic mice. J Exp Med 2000; 191 : 1999–2009. [Google Scholar]
  29. Muso E, Yoshida E, Takeuchi E, et al. Enhanced production of glomerular extracellular matrix in a new mouse strain of high serum IgA ddY mice. Kidney Int 1996; 50 : 1946–57. [Google Scholar]
  30. Zheng F, Kundu GC, Zhang Z, et al. Uteroglobin is essential in preventing immunoglobulin A nephropathy in mice. Nat Med 1999; 5 : 1018–25. [Google Scholar]
  31. Silvain C, Patry C, Launay P, Lehuen A, Monteiro R. Altered expression of monocyte IgA Fc receptors is associated with defective endocytosis in patients with alcoholic cirrhosis. J Immunol 1995; 155 : 1606–18. [Google Scholar]
  32. Grossetête B, Viard JP, Lehuen A, Bach JF, Monteiro R. Impaired Fca receptor expression is linked to increased immunoglobulin A levels and disease progression in HIV-1-infected patients. AIDS 1995; 9 : 229–34. [Google Scholar]
  33. Montenegro V, Chiamolera M, Launay P, Gonçalves CR, Monteiro RC. Impaired expression of IgA Fc receptors (CD89) by blood phagocytic cells in ankylosing spondylitis. J Rheumatol 2000; 27 : 411–7. [Google Scholar]
  34. Westerhuis R, Van Zandbergen G, Verhagen NA, et al. Human mesangial cells in culture and in kidney sections fail to express Fc a receptor (CD89). J Am Soc Nephrol 1999; 10 : 770–8. [Google Scholar]
  35. Barratt J, Greer MR, Pawluczyk IZ, et al. Identification of a novel Fca receptor expressed by human mesangial cells. Kidney Int 2000; 57 : 1936–48. [Google Scholar]
  36. Shibuya A, Sakamoto N, Shimizu Y, et al. Fc a/m receptor mediates endocytosis of IgM-coated microbes. Nat Immunol 2000; 1 : 441–6. [Google Scholar]
  37. McDonald KJ, Cameron AJ, Allen JM, Jardine AG. Expression of Fc a/m receptor by human mesangial cells : a candidate receptor for immune complex deposition in IgA nephropathy. Biochem Biophys Res Commun 2002; 290 : 438–42. [Google Scholar]
  38. Moura IC, Centelles MN, Arcos-Fajardo M, et al. Identification of the transferrin receptor as a novel immunoglobulin (Ig)A1 receptor and its enhanced expression on mesangial cells in IgA nephropathy. J Exp Med 2001; 194 : 417–25. [Google Scholar]
  39. Haddad E, Moura IC, Arcos-Fajardo M, et al. Enhanced expression of the CD71 mesangial IgA1 receptor in Berger’s disease and Henoch-Schönlein nephritis : association between CD71 expression and IgA deposits. J Am Soc Nephrol 2003; 14 : 327–37. [Google Scholar]
  40. Chen A, Chen WP, Sheu LF, Lin CY. Pathogenesis of IgA nephropathy : in vitro activation of human mesangial cells by IgA immune complex leads to cytokine secretion. J Pathol 1994; 173 : 119–26. [Google Scholar]
  41. Van den Dobbelsteen ME, van der Woude FJ, Schroeijers WE, et al. Binding of dimeric and polymeric IgA to rat renal mesangial cells enhances the release of interleukin 6. Kidney Int 1994; 46 : 512–9. [Google Scholar]
  42. Duque N, Gomez-Guerrero C, Egido J. Interaction of IgA with Fca receptors of human mesangial cells activates transcription factor nuclear factor-kB and induces expression and synthesis of monocyte chemoattractant protein-1, IL-8, and IFN-inducible protein 10. J Immunol 1997; 159 : 3474–82. [Google Scholar]
  43. Schena F, Cerullo G, Rossini M, et al. Increased risk of end-stage renal disease in familial IgA nephropathy. J Am Soc Nephrol 2002; 13 : 453–60. [Google Scholar]
  44. Hsu S, Ramirez S, Winn M, Bonventre J, Owen W. Evidence for genetic factors in the development and progression of IgA nephropathy. Kidney Int 2000; 57 : 1818–35. [Google Scholar]
  45. Ghavari A, Scolari F, Schena F, et al. IgA nephropathy, the most common cause of glomerulonephritis, is linked to 6q22-23. Nat Genet 2000; 26 : 354–7. [Google Scholar]

Current usage metrics show cumulative count of Article Views (full-text article views including HTML views, PDF and ePub downloads, according to the available data) and Abstracts Views on Vision4Press platform.

Data correspond to usage on the plateform after 2015. The current usage metrics is available 48-96 hours after online publication and is updated daily on week days.

Initial download of the metrics may take a while.