Open Access
Numéro |
Med Sci (Paris)
Volume 37, Numéro 4, Avril 2021
|
|
---|---|---|
Page(s) | 359 - 365 | |
Section | M/S Revues | |
DOI | https://doi.org/10.1051/medsci/2021032 | |
Publié en ligne | 28 avril 2021 |
- Bankir L, Bichet DG, Morgenthaler NG. Vasopressin: physiology, assessment and osmosensation. J Int Med 2017 ; 282 : 284–297. [Google Scholar]
- Machnik A, Neuhofer W, Jantsch J, et al. Macrophages regulate salt-dependent volume and blood pressure by a vascular endothelial growth factor-C-dependent buffering mechanism. Nat Med 2009 ; 15 : 545–552. [CrossRef] [PubMed] [Google Scholar]
- Cowley AW, Jr, Roman RJ. Control of blood and extracellular volume. Bailliere’s Clinical Endocrinology and Metabolism 1989 ; 3 : 331–369. [Google Scholar]
- Nielsen R, Christensen EI, Birn H. Megalin and cubilin in proximal tubule protein reabsorption: from experimental models to human disease. Kidney Int 2016 ; 89 : 58–67. [CrossRef] [PubMed] [Google Scholar]
- Feraille E, Doucet A. Sodium-potassium-adenosinetriphosphatase-dependent sodium transport in the kidney: hormonal control. Physiol Rev 2001 ; 81 : 345–418. [CrossRef] [PubMed] [Google Scholar]
- Curthoys NP, Moe OW. Proximal tubule function and response to acidosis. Clin J Am Soc Nephrol 2014 ; 9 : 1627–1638. [CrossRef] [PubMed] [Google Scholar]
- Yu ASL. Claudins and the kidney. J Am Soc Nephrol 2015 ; 26 : 11–19. [CrossRef] [PubMed] [Google Scholar]
- Sands JM, Layton HE. Advances in understanding the urine-concentrating mechanism. Ann Rev Physiol 2014 ; 76 : 387–409. [Google Scholar]
- Mount DB. Thick ascending limb of the loop of Henle. Clin J Am Soc Nephrol 2014 ; 9 : 1974–1986. [CrossRef] [PubMed] [Google Scholar]
- Hou J, Renigunta A, Konrad M, et al. Claudin-16 and claudin-19 interact and form a cation-selective tight junction complex. J Clin Invest 2008 ; 118 : 619–628. [PubMed] [Google Scholar]
- Breiderhoff T, Himmerkus N, Stuiver M, et al. Deletion of claudin-10 (Cldn10) in the thick ascending limb impairs paracellular sodium permeability and leads to hypermagnesemia and nephrocalcinosis. Proc Natl Acad Sci USA 2012 ; 109 : 14241–14246. [Google Scholar]
- Milatz S, Himmerkus N, Wulfmeyer VC, et al. Mosaic expression of claudins in thick ascending limbs of Henle results in spatial separation of paracellular Na+ and Mg2+ transport. Proc Natl Acad Sci USA 2017 ; 114 : E219–E227. [Google Scholar]
- Simon DB, Lifton RP. Ion transporter mutations in Gitelman’s and Bartter’s syndromes. Curr Op Nephrol Hypertens 1998 ; 7 : 43–47. [Google Scholar]
- Bongers EM, Shelton LM, Milatz S, et al. A novel hypokaliemic-alkalotic salt loosing tubulopathy in patients with CLDN10 mutations. J Am Soc Nephrol 2017 ; 28 : 3118–3128. [CrossRef] [PubMed] [Google Scholar]
- Hasj-Rabia S, Brideau G, Al-Sarraj Y, el al. Multiplex epithelium dysfunction due to CLDN10 mutation: the HELIX syndrome. Genet Med 2019; 20 : 190–201. [Google Scholar]
- Poulsen SB, Fenton RA. K+ and the renin-angiotensin-aldosterone system: new insights into their role in blood pressure control and hypertension treatment. J Physiol 2019 ; 597 : 4451–4464. [CrossRef] [PubMed] [Google Scholar]
- Staruschenko A. Regulation of transport in the connecting tubule and cortical collecting duct. Comp Physiol 2012 ; 2 : 1541–1584. [Google Scholar]
- Roy A, Al-bataineh MM, Pastor-Soler NM. Collecting duct intercalated cell function and regulation. Clin J Am Soc Nephrol 2015 ; 10 : 305–324. [CrossRef] [PubMed] [Google Scholar]
- Pollak MR, Quaggin SE, Hoenig MP, et al. The glomerulus: the sphere of influence. Clin J Am Soc Nephrol 2014 ; 9 : 1461–1469. [CrossRef] [PubMed] [Google Scholar]
- Bauersachs J, Jaisser F, Toto R. Mineralocorticoid receptor activation and mineralocorticoid receptor antagonist treatment in cardiac and renal diseases. Hypertension 2015 ; 65 : 257–263. [CrossRef] [PubMed] [Google Scholar]
- Fakitsas P, Adam G, Daidie D, et al. Early Aldosterone-Induced gene product regulates the epithelial sodium channel by deubiquitylation. J Am Soc Nephrol 2007 ; 18 : 1084–1092. [CrossRef] [PubMed] [Google Scholar]
- Rotin D, Staub O. Nedd4-2 and the regulation of epithelial sodium transport. Front Physiol 2012 ; 3 : 212. [CrossRef] [PubMed] [Google Scholar]
- Kellenberger S, Gautschi I, Rossier BC, et al. Mutations causing Liddle syndrome reduce sodium-dependent downregulation of the epithelial sodium channel in the Xenopus oocyte expression system. J Clin Invest 1998 ; 101 : 2741–2750. [CrossRef] [PubMed] [Google Scholar]
- May A, Puoti A, Gaeggeler HP, et al. Early effect of aldosterone on the rate of synthesis of the epithelial sodium channel alpha subunit in A6 renal cells. J Am Soc Nephrol 1997 ; 8 : 1813–1822. [CrossRef] [PubMed] [Google Scholar]
- Yu Z, Kong Q, Kone BC. Aldosterone reprograms promoter methylation to regulate αENaC transcription in the collecting cuct. Am J Physiol Renal Physiol 2013 ; 305 : F1006–F1013. [CrossRef] [PubMed] [Google Scholar]
- Wang YB, Leroy V, Maunsbach AB, et al. Sodium transport is modulated by p38 kinase-dependent cross-talk between ENaC and Na, K-ATPase in collecting duct principal cells. J Am Soc Nephrol 2014 ; 25 : 250–259. [CrossRef] [PubMed] [Google Scholar]
- Horisberger JD, Rossier BC. Aldosterone regulation of gene transcription leading to control of ion transport. Hypertension 1992 ; 19 : 221–227. [CrossRef] [PubMed] [Google Scholar]
- Wang WH, Yue P, Sun P, et al. Regulation and function of potassium channels in aldosterone-sensitive distal nephron. Curr Op Nephrol Hypertens 2010 ; 19 : 463–470. [Google Scholar]
- Terker AS, Yarbrough B, Ferdaus MZ, et al. Direct and indirect mineralocorticoid effects determine distal salt transport. J Am Soc Nephrol 2016 ; 27 : 2436–2445. [CrossRef] [PubMed] [Google Scholar]
- Terker AS, Zhang C, McCormick JA, et al. Potassium modulates electrolyte balance and blood pressure through effects on distal cell voltage and chloride. Cell Metab 2015 ; 21 : 39–50. [CrossRef] [PubMed] [Google Scholar]
- Doucet A, Favre G, Deschênes G. Molecular mechanism of edema formation in nephrotic syndrome: therapeutic implications. Pediatric Nephrol 2007 ; 22 : 1983–1990. [Google Scholar]
- Ichikawa I, Rennke HG, Hoyer JR, et al. Role for intrarenal mechanisms in the impaired salt excretion of experimental nephrotic syndrome. J Clin Invest 1983 ; 71 : 91–103. [CrossRef] [PubMed] [Google Scholar]
- Deschenes G, Doucet A, Collecting duct (Na+/K+)-ATPase activity is correlated with urinary sodium excretion in rat nephrotic syndromes. J Am Soc Nephrol 2000 ; 11 : 604–615. [CrossRef] [PubMed] [Google Scholar]
- Lourdel S, Loffing J, Favre G, et al. Hyperaldosteronemia and activation of the epithelial sodium channel are not required for sodium retention in puromycin-induced nephrosis. J Am Soc Nephrol 2005 ; 16 : 3642–3650. [CrossRef] [PubMed] [Google Scholar]
- Vogt B, Favre H. Na+, K+-ATPase activity and hormones in single nephron segments from nephrotic rats. Clin Sci 1991 ; 80 : 599–604. [Google Scholar]
- Apostol E, Ecelbarber CA, Terris J, et al. Reduced renal medullary water channel expression in puromycin aminonucleoside-induced nephrotic syndrome. J Am Soc Nephrol 1997 ; 8 : 15–24. [CrossRef] [PubMed] [Google Scholar]
- Rutkowski JM, Wang ZV, Park ASD, et al. Adiponectin promotes functional recovery after podocyte ablation. J Am Soc Nephrol 2013 ; 24 : 268–282. [CrossRef] [PubMed] [Google Scholar]
- Dizin E, Olivier V, Maire C, et al. Time-course of sodium transport along the nephron in nephrotic syndrome: The role of potassium. FASEB J 2020; 34 : 2408–24. [CrossRef] [PubMed] [Google Scholar]
- Fila M, Brideau G, Morla L, et al. Inhibition of K+ secretion in the distal nephron in nephrotic syndrome: possible role of albuminuria. J Physiol 2011 ; 589 : 3611–3621. [CrossRef] [PubMed] [Google Scholar]
Les statistiques affichées correspondent au cumul d'une part des vues des résumés de l'article et d'autre part des vues et téléchargements de l'article plein-texte (PDF, Full-HTML, ePub... selon les formats disponibles) sur la platefome Vision4Press.
Les statistiques sont disponibles avec un délai de 48 à 96 heures et sont mises à jour quotidiennement en semaine.
Le chargement des statistiques peut être long.