Open Access
Numéro
Med Sci (Paris)
Volume 35, Numéro 3, Mars 2019
Page(s) 236 - 243
Section M/S Revues
DOI https://doi.org/10.1051/medsci/2019036
Publié en ligne 1 avril 2019
  1. Kanner L.. Autistic disturbances of affective contact. Nervous Child 1943 ; 2 : 217–250. [Google Scholar]
  2. Guibert C, Beaud L. Différence entre autisme de Kanner et psychose infantile : déficits d’unité vs d’identité de la situation ?. Psychiatrie de l’Enfant 2005 ; 48 : 391–423. [CrossRef] [Google Scholar]
  3. Wing L, Gould J. Severe impairments of social interaction and associated abnormalities in children: epidemiology and classification. J Autism Dev Disord 1979 ; 9 : 11–29. [Google Scholar]
  4. DSM-III. Diagnostic and statistical manual of mental disorders, 3e ed. Washington DC : American Psychiatric Association, 1980. [Google Scholar]
  5. DSM-V. Diagnostic and statistical manual of mental disorders, 5e ed. Washington DC : American Psychiatric Association, 2013. [Google Scholar]
  6. Bourgeron T.. From the genetic architecture to synaptic plasticity in autism spectrum disorder. Nat Rev Neurosci 2015 ; 16 : 551–563. [CrossRef] [PubMed] [Google Scholar]
  7. Rossignol DA, Genuis SJ, Frye RE. Environmental toxicants and autism spectrum disorders: a systematic review. Transl Psychiatry 2014 ; 4 : e360. [CrossRef] [PubMed] [Google Scholar]
  8. Tran NQV, Miyake K. Neurodevelopmental disorders and environmental toxicants: epigenetics as an underlying mechanism. Int J Genomics 2017 ; 2017 : 7526592. [Google Scholar]
  9. Amiet C.. Diversité clinique de l’autisme : aspects diagnostiques. Perspectives Psy 2007 ; 46 : 228–239. [CrossRef] [EDP Sciences] [Google Scholar]
  10. Ha S, Sohn IJ, Kim N, et al. Characteristics of brains in autism spectrum disorder: Structure, function and connectivity across the lifespan. Exp Neurobiol 2015 ; 24 : 273–284. [Google Scholar]
  11. Herrington JD, Maddox BB, Kerns CM, et al. Amygdala volume differences in autism spectrum disorder are related to anxiety. J Autism Dev Disord 2017 ; 47 : 3682–3691. [Google Scholar]
  12. Langen L, Bos D, Noordermeer SD, et al. Changes in the development of striatum are involved in repetitive behavior in autism. Biol Psychiatry 2014 ; 76 : 405–411. [CrossRef] [PubMed] [Google Scholar]
  13. Donovan AP, Basson M. The neuroanatomy of autism: a developmental perspective. J Anat 2017 ; 230 : 4–15. [Google Scholar]
  14. Roux S, Bossu JL. Le cervelet : des troubles moteurs à l’autisme. Rev Neuropsychol 2016 ; 8 : 182–191. [Google Scholar]
  15. Hull JV, Dokovna LB, Jacokes ZJ, et al. Resting-state functional connectivity in autism spectrum disorders: a review. Front Psychiatry 2017 ; 7 : 205. [CrossRef] [PubMed] [Google Scholar]
  16. Varghese M, Keshav N, Jacot-Descombes S, et al. Autism spectrum disorder: neuropathology and animal models. Acta Neuropathol 2017 ; 134 : 537–566. [CrossRef] [PubMed] [Google Scholar]
  17. Christensen J, Grønborg TK, Sørensen MJ, et al. Prenatal valproate exposure and risk of autism spectrum disorders and childhood autism. JAMA 2013 ; 309 : 1696–1703. [CrossRef] [PubMed] [Google Scholar]
  18. Rodier PM, Ingram JL, Tisdale B, et al. Embryological origin for autism: developmental anomalies of the cranial nerve motor nuclei. J Comp Neurol 1996 ; 370 : 247–261. [CrossRef] [PubMed] [Google Scholar]
  19. Roux S, Bossu JL. Valproic acid and autism spectrum disorder: from clinical observations to animal studies. Curr Trends Neurol 2017 ; 11 : 53–61. [Google Scholar]
  20. Nicolini C, Fahnestock M. The valproic acid-induced rodent model of autism. Exp Neurol 2018 ; 299 : 217–227. [CrossRef] [PubMed] [Google Scholar]
  21. Deckmann I, Schwingel GB, Fontes-Dutra M, et al. Neuroimmune alterations in autism: a translational analysis focusing on the animal model of autism induced by prenatal exposure to valproic acid. Neuroimmunomodulation 2018 ; 29 : 1–15. [Google Scholar]
  22. Yamaguchi H, Hara Y, Ago Y, et al. Environmental enrichment attenuates behavioral abnormalities in valproic acid-exposed autism model mice. Behav Brain Res 2017 ; 333 : 67–73. [CrossRef] [PubMed] [Google Scholar]
  23. Kataoka S, Takuma K, Hara Y, et al. Autism-like behaviours with transient histone hyperacetylation in mice treated prenatally with valproic acid. Int J Neuropsychopharmacol 2013 ; 16 : 91–103. [CrossRef] [PubMed] [Google Scholar]
  24. Howell BW, Smith KM. Synaptic structural protein dysfunction leads to altered excitation inhibition ratios in models of autism spectrum disorder. Pharmacol Res 2018 ; 139 : 207–214. [CrossRef] [PubMed] [Google Scholar]
  25. Roux S, Bailly Y, Bossu JL. Regional and sex-dependent alterations in Purkinje cell density in the valproate mouse model of autism. Neuroreport 2019 ; 30 : 82–88. [CrossRef] [PubMed] [Google Scholar]
  26. Al Sagheer T, Haida O, Balbous A, et al. Motor impairments correlate with social deficits and restricted neuronal loss in an environmental model of autism. Int J Neuropsychopharmacol 2018; 21 : 871–82. [CrossRef] [PubMed] [Google Scholar]
  27. Rubenstein JL, Merzenich MM. Model of autism: increased ratio of excitation/inhibition in key neural systems. Genes Brain Behav 2003 ; 2 : 255–267. [PubMed] [Google Scholar]
  28. Ben-Ari Y.. The GABA excitatory/inhibitory developmental sequence: a personal journey. Neurosci 2014 ; 279 : 187–219. [CrossRef] [Google Scholar]
  29. Tyzio R, Nardou R, Ferrari DC, et al. Oxytocin-mediated GABA inhibition during delivery attenuates autism pathogenesis in rodent offspring. Science 2014 ; 343 : 675–679. [Google Scholar]
  30. Roux S, Lohof A, Ben-Ari Y, et al. Maturation of GABAergic transmission in cerebellar Purkinje Cells is sex dependent and altered in the valproate model of autism. Front Cell Neurosci 2018 ; 12 : 232. [Google Scholar]
  31. Markram K, Markram H. The intense world theory: a unifying theory of the neurobiology of autism. Front Hum Neurosci 2010 ; 4 : 224. [CrossRef] [PubMed] [Google Scholar]
  32. Lemonnier E, Villeneuve N, Sonie S, et al. Effects of bumetanide on neurobehavioral function in children and adolescents with autism spectrum disorders. Transl Psychiatry 2017 ; 7 : e1056. [CrossRef] [PubMed] [Google Scholar]

Les statistiques affichées correspondent au cumul d'une part des vues des résumés de l'article et d'autre part des vues et téléchargements de l'article plein-texte (PDF, Full-HTML, ePub... selon les formats disponibles) sur la platefome Vision4Press.

Les statistiques sont disponibles avec un délai de 48 à 96 heures et sont mises à jour quotidiennement en semaine.

Le chargement des statistiques peut être long.