Free Access
Issue |
Med Sci (Paris)
Volume 22, Number 1, Janvier 2006
|
|
---|---|---|
Page(s) | 54 - 61 | |
Section | M/S revues | |
DOI | https://doi.org/10.1051/medsci/200622154 | |
Published online | 15 January 2006 |
- Feinberg AP. The epigenetics of cancer etiology. Sem Cancer Biol 2004; 14 : 427–32. [Google Scholar]
- Wales MM, Biel MA, El-Deiry W, et al. p53 activates expression of HIC-1, a new candidate tumour suppressor gene on 17p13.3. Nat Med 1995; 1 : 570–7. [Google Scholar]
- Schultz DC, Vanderveer L, Berman DB, et al. Identification of two candidate tumor suppressor genes on chromosome 17p13.3. Cancer Res 1996; 56 : 1997–2002. [Google Scholar]
- Pinte S, Stankovic-Valentin N, Deltour S, et al. The tumor suppressor gene HIC1 (hypermethylated in cancer 1) is a sequence-specific transcriptional repressor : definition of its consensus binding sequence and analysis of its DNA binding and repressive properties. J Biol Chem 2004; 279 : 38313–24. [Google Scholar]
- Deltour S, Pinte S, Guerardel C, et al. The human candidate tumor suppressor gene HIC1 recruits CtBP through a degenerate GLDLSKK motif. Mol Cell Biol 2002; 22 : 4890–901. [Google Scholar]
- Prive GG, Melnick A, Ahmad K, Licht JD. Zinc finger proteins : from atomic contact to cellular function. In : Iuchi S, Kuldell N, eds. Georgetown : Landes Biosciences, 2004. [Google Scholar]
- Deltour S, Guerardel C, Leprince D. Recruitment of SMRT/N-CoR-mSin3A-HDAC repressing complexes is not a general mechanism for BTB/POZ transcriptional repressors : the case of HIC-1 and gammaFBP-B. Proc Natl Acad Sci USA 1999; 96 : 14831–6. [Google Scholar]
- Yang XJ, Gregoire S. Class II histone deacetylases : from sequence to function, regulation, and clinical implication. Mol Cell Biol 2005; 25 : 2873–84. [Google Scholar]
- Yamanaka M, Watanabe M, Yamada Y, et al. Altered methylation of multiple genes in carcinogenesis of the prostate. Int J Cancer 2003; 106 : 382–7. [Google Scholar]
- Kanai Y, Ushijima S, Ochiai A, et al. DNA hypermethylation at the D17S5 locus is associated with gastric carcinogenesis. Cancer Lett 1998; 122 : 135–41. [Google Scholar]
- Pieretti M, Powell DE, Gallion HH, et al. Hypermethylation at a chromosome 17 « hot spot » is a common event in ovarian cancer. Hum Pathol 1995; 26 : 398–401. [Google Scholar]
- Fujii H, Biel MA, Zhou W, et al. Methylation of the HIC-1 candidate tumor suppressor gene in human breast cancer. Oncogene 1998; 16 : 2159–64. [Google Scholar]
- Rood BR, Zhang H, Weitman DM, Cogen PH. Hypermethylation of HIC-1 and 17p allelic loss in medulloblastoma. Cancer Res 2002; 62 : 3794–7. [Google Scholar]
- Chen WY, Zeng X, Carter MG, et al. Heterozygous disruption of Hic1 predisposes mice to a gender-dependent spectrum of malignant tumors. Nat Genet 2003; 33 : 197–202. [Google Scholar]
- Kwabi-Addo B, Giri D, Schmidt K, et al. Haploinsufficiency of the Pten tumor suppressor gene promotes prostate cancer progression. Proc Natl Acad Sci USA 2001; 98 : 11563–8. [Google Scholar]
- DePinho RA. The age of cancer. Nature 2000; 408 : 248–54. [Google Scholar]
- Quon KC, Berns A. Haplo-insufficiency ? Let me count the ways. Genes Dev 2001; 15 : 2917–21. [Google Scholar]
- Wynshaw-Boris A, Gambello MJ. LIS1 and dynein motor function in neuronal migration and development. Genes Dev 2001; 15 : 639–51. [Google Scholar]
- Cardoso C, Leventer RJ, Ward HL, et al. Refinement of a 400-kb critical region allows genotypic differentiation between isolated lissencephaly, Miller-Dieker syndrome, and other phenotypes secondary to deletions of 17p13.3. Am J Hum Genet 2003; 72 : 918–30. [Google Scholar]
- Carter MG, Johns MA, Zeng X, et al. Mice deficient in the candidate tumor suppressor gene Hic1 exhibit developmental defects of structures affected in the Miller-Dieker syndrome. Hum Mol Genet 2000; 9 : 413–9. [Google Scholar]
- Grimm C, Sporle R, Schmid TE, et al. Isolation and embryonic expression of the novel mouse gene Hic1, the homologue of HIC1, a candidate gene for the Miller-Dieker syndrome. Hum Mol Genet 1999; 8 : 697–710. [Google Scholar]
- Chen CM, Behringer RR. Ovca1 regulates cell proliferation, embryonic development, and tumorigenesis. Genes Dev 2004; 18 : 320–32. [Google Scholar]
- Chen W, Cooper TK, Zahnow CA, et al. Epigenetic and genetic loss of Hic1 function accentuates the role of p53 in tumorigenesis. Cancer Cell 2004; 6 : 387–98. [Google Scholar]
- Britschgi C, Rizzi M, Grob TJ, et al. Identification of the p53 family-responsive element in the promoter region of the tumor suppressor gene hypermethylated in cancer 1. Oncogene 2005; november 21 (online). [Google Scholar]
- Guerardel C, Deltour S, Pinte S, et al. Identification in the human candidate tumor suppressor gene HIC-1 of a new major alternative TATA-less promoter positively regulated by p53. J Biol Chem 2001; 276 : 3078–89. [Google Scholar]
- Chen W, Wamng DH, Yen RC, et al. Tumor suppressor HIC1 directly regulates SIRT1 to modulate p53 dependent DNA damage responses. Cell 2005; 123 : 437–48. [Google Scholar]
- Vaziri H, Dessain SK, Ng-Eaton E, et al. hSIR2(SIRT1) functions as an NAD-dependent p53 deacetylase. Cell 2001; 107 : 149–59. [Google Scholar]
- Senawong T, Peterson VJ, Leid M. BCL11A-dependent recruitment of SIRT1 to a promoter template in mammalian cells results in histone deacetylation and transcriptional repression. Arch Biochem Biophys 2005; 434 : 316–25. [Google Scholar]
- Chen WY, Baylin SB. Inactivation of tumor suppressor genes : choice between genetic and epigenetic routes. Cell Cycle 2005; 4 : 10–2. [Google Scholar]
- Nicoll G, Crichton DN, McDowell HE, et al. Expression of the hypermethylated in cancer gene (HIC-1) is associated with good outcome in human breast cancer. Br J Cancer 2001; 85 : 1878–82. [Google Scholar]
Current usage metrics show cumulative count of Article Views (full-text article views including HTML views, PDF and ePub downloads, according to the available data) and Abstracts Views on Vision4Press platform.
Data correspond to usage on the plateform after 2015. The current usage metrics is available 48-96 hours after online publication and is updated daily on week days.
Initial download of the metrics may take a while.