Open Access
Med Sci (Paris)
Volume 37, Number 10, Octobre 2021
Page(s) 848 - 850
Section Le Magazine
Published online 14 October 2021
  1. Borie AM, Dromard Y, Guillon G, et al. Correction of vasopressin deficit in the lateral septum ameliorates social deficits of mouse autism model. J Clin Invest 2021; 131 : e144450. [Google Scholar]
  2. Oztan O, Garner JP, Partap S, et al. Cerebrospinal fluid vasopressin and symptom severity in children with autism. Ann Neurol 2018 ; 84 : 611–615. [CrossRef] [PubMed] [Google Scholar]
  3. Parker KJ, Oztan O, Libove RA, et al. A randomized placebo-controlled pilot trial shows that intranasal vasopressin improves social deficits in children with autism. Sci Transl Med 2019; eaau7356. [CrossRef] [PubMed] [Google Scholar]
  4. Bolognani F, del Valle Rubido M, Squassante L, et al. A phase 2 clinical trial of a vasopressin V1a receptor antagonist shows improved adaptive behaviors in men with autism spectrum disorder. Sci Transl Med 2019; eaat7838. [CrossRef] [PubMed] [Google Scholar]
  5. Tauber M, Boulanouar K, Diene G, et al. The use of oxytocin to improve feeding and social skills in infants with Prader-Willi syndrome. Pediatrics 2017 ; 139 : e20162976. [CrossRef] [PubMed] [Google Scholar]
  6. Bernaerts S, Boets B, Bosmans G, et al. Behavioral effects of multiple-dose oxytocin treatment in autism: a randomized, placebo-controlled trial with long-term follow-up. Mol Autism 2020; 11 : 6. [CrossRef] [PubMed] [Google Scholar]
  7. Schaaf CP, Gonzalez-Garay ML, Xia F, et al. Truncating mutations of MAGEL2 cause Prader-Willi phenotypes and autism. Nat Genet 2013 ; 45 : 1405–1408. [CrossRef] [PubMed] [Google Scholar]
  8. Meziane H, Fabienne S, Sylvian B, et al. An early postnatal oxytocin treatment prevents social and learning deficits in adult mice deficient for Magel2, a gene involved in Prader-Willi syndrome and autism. Biol Psy 2015 ; 78 : 85–94. [Google Scholar]
  9. Schaller F, Watrin F, Sturny R, et al. A single postnatal injection of oxytocin rescues the lethal feeding behaviour in mouse newborns deficient for the imprinted Magel2 gene. Hum Mol Genet 2010 ; 19 : 4895–4905. [CrossRef] [PubMed] [Google Scholar]
  10. Rigney N, Whylings J, Mieda M, et al. Sexually dimorphic vasopressin cells modulate social investigation and communication in sex-specific ways. eNeuro 2019; 6 : eneuro.0415-18.2019. [Google Scholar]
  11. Chini B, Manning M, Guillon G. Affinity and efficacy of selective agonists and antagonists for vasopressin and oxytocin receptors: an ‘easy guide’ to receptor pharmacology. Prog Brain Res 2008 ; 170 : 513–517. [CrossRef] [PubMed] [Google Scholar]
  12. Peñagarikano O, Lázaro MT, Lu X-H, et al. Exogenous and evoked oxytocin restores social behavior in the Cntnap2 mouse model of autism. Sci Transl Med 2015; 21 : 271ra8. [Google Scholar]
  13. Bales KL, Plotsky PM, Young LJ, et al. Neonatal oxytocin manipulations have long-lasting, sexually dimorphic effects on vasopressin receptors. Neuroscience 2007 ; 144 : 38–45. [CrossRef] [PubMed] [Google Scholar]

Current usage metrics show cumulative count of Article Views (full-text article views including HTML views, PDF and ePub downloads, according to the available data) and Abstracts Views on Vision4Press platform.

Data correspond to usage on the plateform after 2015. The current usage metrics is available 48-96 hours after online publication and is updated daily on week days.

Initial download of the metrics may take a while.