Free Access
Issue |
Med Sci (Paris)
Volume 21, Number 2, Février 2005
|
|
---|---|---|
Page(s) | 162 - 169 | |
Section | M/S revues | |
DOI | https://doi.org/10.1051/medsci/2005212162 | |
Published online | 15 February 2005 |
- Arroyo EJ, Scherer SS. On the molecular architecture of myelinated fibers. Histochem Cell Biol 2000; 113 : 1–18. [Google Scholar]
- Filbin MT, Tennekoon GI. The role of complex carbohydrates in adhesion of the myelin protein, P0. Neuron 1991; 7 : 845–55. [Google Scholar]
- Tetzlaff W. Tight junction contact events and temporary gap junctions in the sciatic nerve fibres of the chicken during Wallerian degeneration and subsequent regeneration. J Neurocytol 1982; 11 : 839–58. [Google Scholar]
- Fannon AM, Sherman DL, Ilyina-Gragerova G, et al. Novel E-cadherin-mediated adhesion in peripheral nerve : Schwann cell architecture is stabilized by autotypic adherens junctions. J Cell Biol 1995; 129 : 189–202. [Google Scholar]
- Gow A, Southwood CM, Li JS, et al. CNS myelin and Sertoli cell tight junction strands are absent in Osp/claudin-11 null mice. Cell 1999; 99 : 649–59. [Google Scholar]
- Tsukita S, Furuse M. Claudin-based barrier in simple and stratified cellular sheets. Curr Opin Cell Biol 2002; 14 : 531–6. [Google Scholar]
- Poliak S, Matlis S, Ullmer C, et al. Distinct claudins and associated PDZ proteins form different autotypic tight junctions in myelinating Schwann cells. J Cell Biol 2002; 159 : 361–72. [Google Scholar]
- Nicholson BJ. Gap junctions : from cell to molecule. J Cell Sci 2003; 116 : 4479–81. [Google Scholar]
- Balice-Gordon RJ, Bone LJ, Scherer SS. Functional gap junctions in the schwann cell myelin sheath. J Cell Biol 1998; 142 : 1095–104. [Google Scholar]
- Altevogt BM, Kleopa KA, Postma FR, et al. Connexin29 is uniquely distributed within myelinating glial cells of the central and peripheral nervous systems. J Neurosci 2002; 22 : 6458–70. [Google Scholar]
- Scherer SS, Deschenes SM, Xu YT, et al. Connexin32 is a myelin-related protein in the PNS and CNS. J Neurosci 1995; 15 : 8281–94. [Google Scholar]
- Suter U, Scherer SS. Disease mechanisms in inherited neuropathies. Nat Rev Neurosci 2003; 4 : 714–26. [Google Scholar]
- Berger P, Young P, Suter U. Molecular cell biology of Charcot-Marie-Tooth disease. Neurogenetics 2002; 4 : 1–15. [Google Scholar]
- Young P, Boussadia O, Berger P, et al. E-cadherin is required for the correct formation of autotypic adherens junctions of the outer mesaxon but not for the integrity of myelinated fibers of peripheral nerves. Mol Cell Neurosci 2002; 21 : 341–51. [Google Scholar]
- Yamada H, Denzer AJ, Hori H, et al. Dystroglycan is a dual receptor for agrin and laminin-2 in Schwann cell membrane. J Biol Chem 1996; 271 : 23418–23. [Google Scholar]
- Sherman DL, Fabrizi C, Gillespie CS, et al. Specific disruption of a Schwann cell dystrophin-related protein complex in a demyelinating neuropathy. Neuron 2001; 30 : 677–87. [Google Scholar]
- Gillespie CS, Sherman DL, Fleetwood-Walker SM, et al. Peripheral demyelination and neuropathic pain behavior in periaxin-deficient mice. Neuron 2000; 26 : 523–31. [Google Scholar]
- Boerkoel CF, Takashima H, Stankiewicz P, et al. Periaxin mutations cause recessive Dejerine-Sottas neuropathy. Am J Hum Genet 2001; 68 : 325–33. [Google Scholar]
- Guilbot A, Williams A, Ravise N, et al. A mutation in periaxin is responsible for CMT4F, an autosomal recessive form of Charcot-Marie-Tooth disease. Hum Mol Genet 2001; 10 : 415–21. [Google Scholar]
- Saito F, Moore SA, Barresi R, et al. Unique role of dystroglycan in peripheral nerve myelination, nodal structure, and sodium channel stabilization. Neuron 2003; 38 : 747–58. [Google Scholar]
- Rambukkana A. Mycobacterium leprae-induced demyelination : a model for early degeneration. Curr Opin Immunol 2004; 16 : 511–8. [Google Scholar]
- Matsumura K, Yamada H, Saito F, et al. Peripheral nerve involvement in merosin-deficient cognenital muscular dystrophy and dy mouse. Neuromusc. Disorders 1997; 7 :7–12. [Google Scholar]
- Salzer JL. Polarized domains of myelinated axons. Neuron 2003; 40 : 297–318. [Google Scholar]
- Goutebroze L, Carnaud M, Denisenko N, et al. Syndecan-3 and syndecan-4 are enriched in Schwann cell perinodal processes. BMC Neurosci 2003; 4 : 29. [Google Scholar]
- Bhat MA, Rios JC, Lu Y, et al. Axon-glia interactions and the domain organization of myelinated axons require neurexin IV/Caspr/Paranodin. Neuron 2001; 30 : 369–83. [Google Scholar]
- Boyle ME, Berglund EO, Murai KK, et al. Contactin orchestrates assembly of the septate-like junctions at the paranode in myelinated peripheral nerve. Neuron 2001; 30 : 385–97. [Google Scholar]
- Dupree JL, Girault JA, Popko B. Axo-glial interactions regulate the localization of axonal paranodal proteins. J Cell Biol 1999; 147 : 1145–52. [Google Scholar]
- Ishibashi T, Dupree JL, Ikenaka K, et al. A myelin galactolipid, sulfatide, is essential for maintenance of ion channels on myelinated axon but not essential for initial cluster formation. J Neurosci 2002; 22 : 6507–14. [Google Scholar]
- Poliak S, Gollan L, Salomon D, et al. Localization of Caspr2 in myelinated nerves depends on axon-glia interactions and the generation of barriers along the axon. J Neurosci 2001; 21 : 7568–75. [Google Scholar]
- Poliak S, Salomon D, Elhanany H, et al. Juxtaparanodal clustering of Shaker-like K+ channels in myelinated axons depends on Caspr2 and TAG-1. J Cell Biol 2003; 162 : 1149–60. [Google Scholar]
- Traka M, Goutebroze L, Denisenko N, et al. Association of TAG-1 with Caspr2 is essential for the molecular organization of juxtaparanodal regions of myelinated fibers. J Cell Biol 2003; 162 : 1161–72. [Google Scholar]
Current usage metrics show cumulative count of Article Views (full-text article views including HTML views, PDF and ePub downloads, according to the available data) and Abstracts Views on Vision4Press platform.
Data correspond to usage on the plateform after 2015. The current usage metrics is available 48-96 hours after online publication and is updated daily on week days.
Initial download of the metrics may take a while.