EDP Sciences logo
Subscriber Authentication Point
Search
Menu
Home All issues Volume 20 / No 11 (Novembre 2004) Med Sci (Paris), 20 11 (2004) 1014-1019 References
Free Access
Issue
Med Sci (Paris)
Volume 20, Number 11, Novembre 2004
Page(s) 1014 - 1019
Section M/S revues
DOI https://doi.org/10.1051/medsci/200420111014
Published online 15 November 2004
  1. Gisselbrecht S. Oncogenes and leukemia : history and perspectives. Med Sci (Paris) 2003; 19 : 201–10. [Google Scholar]
  2. Boyd KE, Farnham PJ. Identification of target genes of oncogenic transcription factors. Proc Soc Exp Biol Med 1999; 222 : 9–28. [Google Scholar]
  3. Look AT. Oncogenic transcription factors in the human acute leukemias. Science 1997; 278 : 1059–64. [Google Scholar]
  4. Dittmer J, Nordheim A. Ets transcription factors and human disease. Biochim Biophys Acta 1998; 1377 : F1–11. [Google Scholar]
  5. Galiana-Arnoux D, Lejeune F, Gesnel MC, et al. The CD44 alternative v9 exon contains a splicing enhancer responsive to the SR proteins 9G8, ASF/SF2, and SRp20. J Biol Chem 2003; 278 : 32943–53. [Google Scholar]
  6. Caballero OL, de Souza SJ, Brentani RR, Simpson AJ. Alternative spliced transcripts as cancer markers. Dis Markers 2001; 17 : 67–75. [Google Scholar]
  7. Knoop LL, Baker SJ. The splicing factor U1C represses EWS/FLI-mediated transactivation. J Biol Chem 2000; 275 : 24865–71. [Google Scholar]
  8. Poleev A, Hartmann A, Stamm S. A trans-acting factor, isolated by the three-hybrid system, that influences alternative splicing of the amyloid precursor protein minigene. Eur J Biochem 2000; 267 : 4002–10. [Google Scholar]
  9. Yang L, Embree LJ, Hickstein DD. TLS-ERG leukemia fusion protein inhibits RNA splicing mediated by serine-arginine proteins. Mol Cell Biol 2000; 20 : 3345–54. [Google Scholar]
  10. Knoop LL, Baker SJ. EWS/FLI alters 5’-splice site selection. J Biol Chem 2001; 276 : 22317–22. [Google Scholar]
  11. Yang L, Chansky HA, Hickstein DD. EWS.Fli-1 fusion protein interacts with hyperphosphorylated RNA polymerase II and interferes with serine-arginine protein-mediated RNA splicing. J Biol Chem 2000; 275 : 37612–8. [Google Scholar]
  12. Chansky HA, Hu M, Hickstein DD, Yang L. Oncogenic TLS/ERG and EWS/Fli-1 fusion proteins inhibit RNA splicing mediated by YB-1 protein. Cancer Res 2001; 61 : 3586–90. [Google Scholar]
  13. Moreau-Gachelin F, Wendling F, Molina T, et al. Spi-1/PU.1 transgenic mice develop multistep erythroleukemias. Mol Cell Biol 1996; 16 : 2453–63. [Google Scholar]
  14. Starck J, Doubeikovski A, Sarrazin S, et al. Spi-1/PU.1 is a positive regulator of the Fli-1 gene involved in inhibition of erythroid differentiation in friend erythroleukemic cell lines. Mol Cell Biol 1999; 19 : 121–35. [Google Scholar]
  15. Cantor AB, Orkin SH. Transcriptional regulation of erythropoiesis : an affair involving multiple partners. Oncogene 2002; 21 : 3368–76. [Google Scholar]
  16. Hallier M, Tavitian A, Moreau-Gachelin F. The transcription factor Spi-1/PU.1 binds RNA and interferes with the RNA-binding protein p54nrb. J Biol Chem 1996; 271 : 11177–81. [Google Scholar]
  17. Hallier M, Lerga A, Barnache S, et al. The transcription factor Spi-1/PU.1 interacts with the potential splicing factor TLS. J Biol Chem 1998; 273 : 4838–42. [Google Scholar]
  18. Deguillien M, Huang SC, Morinière M, et al. Multiple cis elements regulate an alternative splicing event at 4.1R pre-mRNA during erythroid differentiation. Blood 2001; 98 : 3809–16. [Google Scholar]
  19. Théoleyre O, Deguillien M, Morinière M, et al. Spi-1/PU.1 but not Fli-1 inhibits erythroid-specific alternative splicing of 4.1R pre-mRNA in murine erythroleukemia cells. Oncogene 2004; 23 : 920–7. [Google Scholar]
  20. Orphanides G, Reinberg D. A unified theory of gene expression. Cell 2002; 108 : 439–51. [Google Scholar]
  21. Nogues G, Kadener S, Cramer P, et al. Transcriptional activators differ in their abilities to control alternative splicing. J Biol Chem 2002; 277 : 43110–4. [Google Scholar]
  22. Kadener S, Fededa JP, Rosbash M, Kornblihtt AR. Regulation of alternative splicing by a transcriptional enhancer through RNA pol II elongation. Proc Natl Acad Sci USA 2002; 99 : 8185–90. [Google Scholar]
  23. Furger A, O’Sullivan JM, Binnie A, et al. Promoter proximal splice sites enhance transcription. Genes Dev 2002; 16 : 2792–9. [Google Scholar]
  24. Kwek KY, Murphy S, Furger A, et al. U1 snRNA associates with TFIIH and regulates transcriptional initiation. Nat Struct Biol 2002; 9 : 800–5. [Google Scholar]
  25. Ge H, Si Y, Wolffe AP. A novel transcriptional coactivator, p52, functionally interacts with the essential splicing factor ASF/SF2. Mol Cell 1998; 2 : 751–9. [Google Scholar]
  26. Fong YW, Zhou Q. Stimulatory effect of splicing factors on transcriptional elongation. Nature 2001; 414 : 929–33. [Google Scholar]
  27. Tian H. RNA ligands generated against complex nuclear targets indicate a role for U1 snRNP in co-ordinating transcription and RNA splicing. FEBS Lett 2001; 509 : 282–6. [Google Scholar]
  28. Lai MC, Teh BH, Tarn WY. A human papillomavirus E2 transcriptional activator. The interactions with cellular splicing factors and potential function in pre-mRNA processing. J Biol Chem 1999; 274 : 11832–41. [Google Scholar]

Current usage metrics show cumulative count of Article Views (full-text article views including HTML views, PDF and ePub downloads, according to the available data) and Abstracts Views on Vision4Press platform.

Data correspond to usage on the plateform after 2015. The current usage metrics is available 48-96 hours after online publication and is updated daily on week days.

Initial download of the metrics may take a while.