Free Access
Issue
Med Sci (Paris)
Volume 19, Number 8-9, Août-Septembre 2003
Page(s) 847 - 853
Section M/S Revues
DOI https://doi.org/10.1051/medsci/20031989847
Published online 15 August 2003
  1. Kahn SE. The importance of β-cell failure in the development and progression of type 2 diabetes. J Clin Endocrinol Metab 2001; 86; 4047–8. [Google Scholar]
  2. Maechler P, Wolheim CB. Mitochondrial function in normal and diabetic β- cells. Nature 2001; 414; 807–12. [Google Scholar]
  3. Neel JV. Diabetes mellitus ; a «thrifty»genotype rendered detrimental by «progress»? Am J Hum Genet 1962; 14; 353–62. [Google Scholar]
  4. Goto Y, Kakizaki M, Masaki N. Spontaneous diabetes produced by selective breeding of normal Wistar rats. Proc Jpn Acad 1975; 51; 80–5. [Google Scholar]
  5. Portha B, Giroix MH, Serradas P, et al. β-cell function and viability in the spontaneously diabetic GK rat. Information from the GK/Par colony. Diabetes 2001; 50; 89–93. [Google Scholar]
  6. Jonas JC, Sharma A, Hasenkamp W, et al. Chronic hyperglycemia triggers loss of pancreatic β cell differentiation in an animal model of diabetes. J Biol Chem 1999; 274; 14112–21. [Google Scholar]
  7. Barker DJ. The fetal origins of diseases of old age. Eur J Clin Nutr 1992; 46; S3–S6. [Google Scholar]
  8. Patel MS, Srinivasan M. metabolic programming; causes and consequences. J Biol Chem 2002; 277; 1629–32. [Google Scholar]
  9. Blondeau B, Garofano A, Czernichow P, Bréant B. Age-dependent inability of the endocrine pancreas to adapt to pregnancy; a long-term consequence of perinatal malnutrition in the rat. Endocrinology 1999; 140; 4208–13. [Google Scholar]
  10. Bertin E, Gangnerau MN, Bellon G, Bailbé D, Arbelot De Vacqueur A, Portha B. Development of β-cell mass in fetuses of rats deprived of protein and/or energy in last trimester of pregnancy. Am J Physiol 2002; 283; R623–R630. [Google Scholar]
  11. Bihoreau MT, Ktorza A, Kinebanyan MF, Picon L. Impaired glucose homeostasis in adult rats from hyperglycemic mothers. Diabetes 1986; 35; 979–84. [Google Scholar]
  12. Kwong WY, Wild AE, Roberts P, Willis AC, Fleming TP. Maternal undernutrition during the preimplantation period of rat development causes blastocyst abnormalities and programming of postnatal hypertension. Development 2000; 127 ; 4195–202. [Google Scholar]
  13. Gauguier D, Bihoreau MT, Ktorza A, Berthault MF, Picon L. Inheritance of diabetes mellitus as consequence of gestational hyperglycemia in rats. Diabetes 1990; 39; 734–9. [Google Scholar]
  14. Waterland RA, Garza C. Potential mechanisms of metabolic imprinting that lead to chronic disease. Am J Clin Nutr 1999; 69; 179- 97. [Google Scholar]
  15. Kierszenbaum AL. Genomic imprinting and epigenetic reprogramming; unearthing the garden of forking paths. Mol Reprod Dev 2002; 63; 269–72. [Google Scholar]
  16. Jacob RA. Folate, DNA methylation, and gene expression; factors of nature and nurture. Am J Clin Nutr 2000; 72; 903–4. [Google Scholar]
  17. Gauguier D, Froguel P, Parent V, et al. Chromosomal mapping of genetic loci associated with non-insulin dependent diabetes in the GK rat. Nat Genet 1996; 12; 38–43. [Google Scholar]

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