Med Sci (Paris)
Volume 18, Number 12, Décembre 2002
|Page(s)||1178 - 1180|
|Section||Le Magazine : Nouvelles|
|Published online||15 December 2002|
- Gruber SB, Ellis NA, Rennert G, et al. BLM heterozygosity and the risk of colorectal cancer. Science 2002; 297: 2013. [Google Scholar]
- Goss KH, Risinger MA, Kordich JJ, et al. Enhanced tumor formation in mice heterozygous for Blm mutation. Science 2002; 297: 2051–3. [Google Scholar]
- German J. Bloom’s syndrome XX. The first 100 cancers. Cancer Genet Cytogenet 1997; 93: 100–6. [Google Scholar]
- German J. Bloom syndrome : a mendelian prototype of somatic mutational disease. Medicine (Baltimore) 1993; 72: 393–406. [Google Scholar]
- Ellis NA, Groden J, Ye TZ, et al. The Bloom’s syndrome gene product is homologous to RecQ helicases. Cell 1995; 83: 655–66. [Google Scholar]
- Spring K, Ahangari F, Scott SP, et al. Mice heterozygous for mutation in Atm, the gene involved in ataxiatelangiectasia, have heightened susceptibility to cancer. Nat Genet 2002; 32: 185–90. [Google Scholar]
- Karow JK, Chakraverty RK, Hickson ID. The Bloom’s syndrome gene product is a 3’-5’ DNA helicase. J Biol Chem 1997; 272: 30611–4. [Google Scholar]
- Wang Y, Cortez D, Yazdi P, Neff N, Elledge SJ, Qin J. BASC, a super complex of BRCA1-associated proteins involved in the recognition and repair of aberrant DNA structures. Genes Dev 2000; 14: 927–39. [Google Scholar]
- Karow JK, Constantinou A, Li JL, West SC, Hickson ID. The Bloom’s syndrome gene product promotes branch migration of holliday junctions. Proc Natl Acad Sci USA 2000; 97: 6504–8. [Google Scholar]
- Huber MD, Lee DC, Maizels N. G4 DNA unwinding by BLM and Sgs1p : substrate specificity and substrate specific inhibition. Nucleic Acids Res 2002; 30: 3954–61. [Google Scholar]
- Ababou M, Dutertre S, Lecluse Y, Onclercq R, Chatton B, Amor-Gueret M. ATM-dependent phosphorylation and accumulation of endogenous BLM protein in response to ionizing radiation. Oncogene 2000; 19: 5955–63. [Google Scholar]
- Dutertre S, Sekhri R, Tintignac LA, et al. Dephosphorylation and subcellular compartment change of the mitotic Bloom’s syndrome DNA helicase in response to ionizing radiation. J Biol Chem 2002; 277: 6280–6. [Google Scholar]
- Luo G, Santoro IM, McDaniel LD, et al. Cancer predisposition caused by elevated mitotic recombination in Bloom mice. Nat Genet 2000; 25: 424–9. [Google Scholar]
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