Open Access
Numéro
Med Sci (Paris)
Volume 40, Numéro 3, Mars 2024
Page(s) 275 - 282
Section M/S Revues
DOI https://doi.org/10.1051/medsci/2024011
Publié en ligne 22 mars 2024
  1. Hayflick L.. The limited in vitro lifetime of human diploid cell strains. Exp Cell Res 1965 ; 37 : 614–636. [CrossRef] [PubMed] [Google Scholar]
  2. Hayflick L, Moorhead PS. The serial cultivation of human diploid cell strains. Exp Cell Res 1961 ; 25 : 585–621. [CrossRef] [PubMed] [Google Scholar]
  3. Ramirez RD, Morales CP, Herbert BS, et al. Putative telomere-independent mechanisms of replicative aging reflect inadequate growth conditions. Genes Dev 2001 ; 15 : 398–403. [CrossRef] [PubMed] [Google Scholar]
  4. Chaib S, Tchkonia T, Kirkland JL. Cellular senescence and senolytics : the path to the clinic. Nat Med 2022; 28 : 1 556–68. [Google Scholar]
  5. Calcinotto A, Kohli J, Zagato E, et al. Cellular Senescence : Aging, Cancer, and Injury. Physiol Rev 2019 ; 99(1): 047–078. [Google Scholar]
  6. Bousset L, Gil J. Targeting senescence as an anticancer therapy. Mol Oncol 2022; 16 : 3 855–80. [Google Scholar]
  7. Abbadie C, Pluquet O, Pourtier A. Epithelial cell senescence : an adaptive response to pre-carcinogenic stresses ?. Cell Mol Life Sci 2017 ; 74 : 4471–4509. [CrossRef] [PubMed] [Google Scholar]
  8. Pluquet O, Pourtier A, Abbadie C. The unfolded protein response and cellular senescence. A review in the theme : cellular mechanisms of endoplasmic reticulum stress signaling in health and disease. Am J Physiol Cell Physiol 2014 ; 308 : C415–C425. [Google Scholar]
  9. Malaquin N, Martinez A, Rodier F. Keeping the senescence secretome under control : Molecular reins on the senescence-associated secretory phenotype. Exp Gerontol 2016 ; 82 : 39–49. [CrossRef] [PubMed] [Google Scholar]
  10. Di Micco R, Krizhanovsky V, Baker D, et al. Cellular senescence in ageing : from mechanisms to therapeutic opportunities. Nat Rev Mol Cell Biol 2021; 22 : 75–95. [CrossRef] [PubMed] [Google Scholar]
  11. Hu L, Li H, Zi M, et al. Why Senescent Cells Are Resistant to Apoptosis : An Insight for Senolytic Development. Front Cell Dev Biol 2022; 10 : 822 816. [CrossRef] [Google Scholar]
  12. Gao J, Pickett HA. Targeting telomeres : advances in telomere maintenance mechanism-specific cancer therapies. Nat Rev Cancer 2022; 22 : 515–32. [CrossRef] [PubMed] [Google Scholar]
  13. Goy E, Abbadie C. Sénescence et cancer : double jeu. Med Sci (Paris) 2018 ; 34 : 223–230. [CrossRef] [EDP Sciences] [PubMed] [Google Scholar]
  14. Hernandez-Segura A, Nehme J, Demaria M. Hallmarks of Cellular Senescence. Trends Cell Biol 2018 ; 28 : 436–453. [CrossRef] [Google Scholar]
  15. Gosselin K, Deruy E, Martien S, et al. Senescent keratinocytes die by autophagic programmed cell death. Am J Pathol 2009 ; 174 : 423–435. [CrossRef] [PubMed] [Google Scholar]
  16. Deruy E, Gosselin K, Vercamer C, et al. MnSOD upregulation induces autophagic programmed cell death in senescent keratinocytes. PLoS One 2010 ; 5 : e12712. [Google Scholar]
  17. Deruy E, Nassour J, Martin N, et al. Level of macroautophagy drives senescent keratinocytes into cell death or neoplastic evasion. Cell Death Dis 2014 ; 5 : e1577. [CrossRef] [PubMed] [Google Scholar]
  18. Nassour J, Martien S, Martin N, et al. Defective DNA single-strand break repair is responsible for senescence and neoplastic escape of epithelial cells. Nat Commun 2016 ; 7(10): 399. [CrossRef] [Google Scholar]
  19. Goy E, Tomezak M, Facchin C, et al. The out-of-field dose in radiation therapy induces delayed tumorigenesis by senescence evasion. eLife 2022; 11 : e67190. [CrossRef] [PubMed] [Google Scholar]
  20. Goy E, Martin N, Drullion C, et al. Flow Cytometry-based Method for Efficient Sorting of Senescent Cells. BIO-Protoc. 2023; 13. [Google Scholar]
  21. Flor AC, Doshi AP, Kron SJ. Modulation of therapy-induced senescence by reactive lipid aldehydes. Cell Death Discov 2016 ; 2(16): 045. [Google Scholar]
  22. Debacq-Chainiaux F, Erusalimsky JD, Campisi J, et al. Protocols to detect senescence-associated beta-galactosidase (SA-betagal) activity, a biomarker of senescent cells in culture and in vivo. Nat Protoc 2009 ; 4(1): 798–806. [Google Scholar]
  23. Kim KM, Noh JH, Bodogai M, et al. Identification of senescent cell surface targetable protein DPP4. Genes Dev 2017 ; 31 : 1529–1534. [CrossRef] [PubMed] [Google Scholar]
  24. Admasu TD, Kim K, Rae M, et al. Selective ablation of primary and paracrine senescent cells by targeting iron dyshomeostasis. Cell Rep 2023; 42 : 112058. [CrossRef] [PubMed] [Google Scholar]
  25. Verhulst E, Garnier D, De Meester I, et al. Validating Cell Surface Proteases as Drug Targets for Cancer Therapy : What Do We Know, and Where Do We Go ? Cancers 2022; 14 : 624. [CrossRef] [PubMed] [Google Scholar]
  26. Greenberg SB, Grove GL, Cristofalo VJ. Cell size in aging monolayer cultures. In Vitro 1977 ; 13 : 297–300. [CrossRef] [PubMed] [Google Scholar]
  27. Terman A, Brunk UT. Oxidative Stress, Accumulation of Biological “Garbage”, and Aging. Antioxid Redox Signal 2006 ; 8 : 197–204. [CrossRef] [PubMed] [Google Scholar]
  28. Yin D.. Biochemical basis of lipofuscin, ceroid, and age pigment-like fluorophores. Free Radic Biol Med 1996 ; 21 : 871–888. [CrossRef] [PubMed] [Google Scholar]
  29. Pantelis P, Theocharous G, Lagopati N, et al. The Dual Role of Oxidative-Stress-Induced Autophagy in Cellular Senescence : Comprehension and Therapeutic Approaches. Antioxidants 2023; 12 : 169. [CrossRef] [PubMed] [Google Scholar]
  30. Gosselin K, Martien S, Pourtier A, et al. Senescence-associated oxidative DNA damage promotes the generation of neoplastic cells. Cancer Res 2009 ; 69 : 7917–7925. [CrossRef] [PubMed] [Google Scholar]
  31. Bernard D, Gosselin K, Monte D, et al. Involvement of Rel/NF-kappaB transcription factors in keratinocyte senescence. Cancer Res 2004 ; 64 : 472–481. [CrossRef] [PubMed] [Google Scholar]

Les statistiques affichées correspondent au cumul d'une part des vues des résumés de l'article et d'autre part des vues et téléchargements de l'article plein-texte (PDF, Full-HTML, ePub... selon les formats disponibles) sur la platefome Vision4Press.

Les statistiques sont disponibles avec un délai de 48 à 96 heures et sont mises à jour quotidiennement en semaine.

Le chargement des statistiques peut être long.