Le rôle des microvésicules dans l’infection palustre

Karell G. Pellé; Ambroise D. Ahouidi; Pierre-Yves Mantel

doi:10.1051/medsci/20132911010

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Volume 29 / Numéro 11 (Novembre 2013)

Med Sci (Paris), 29 11 (2013) 960-962

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Numéro		Med Sci (Paris) Volume 29, Numéro 11, Novembre 2013


Page(s)		960 - 962
Section		Nouvelles
DOI		https://doi.org/10.1051/medsci/20132911010
Publié en ligne		20 novembre 2013

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Couper KN, Barnes T, Hafalla JC, et al. Parasite-derived plasma microparticles contribute significantly to malaria infection-induced inflammation through potent macrophage stimulation. PLoS Pathog 2010 ; 6 : e1000744. [CrossRef] [PubMed] [Google Scholar]
Mantel PY, Hoang AN, Goldowitz I, et al. Malaria-infected erythrocyte-derived microvesicles mediate cellular communication within the parasite population and with the host immune system. Cell Host Microbe 2013 ; 13 : 521–534. [CrossRef] [PubMed] [Google Scholar]
Regev-Rudzki N, Wilson DW, Carvalho TG, et al. Cell-cell communication between malaria-infected red blood cells via exosome-like vesicles. Cell 2013 ; 153 : 1120–1133. [CrossRef] [PubMed] [Google Scholar]
Baunaure F, Langsley G. Trafic protéique dans le globule rouge infecté par Plasmodium. Med Sci (Paris) 2005 ; 21 : 523–529. [CrossRef] [EDP Sciences] [PubMed] [Google Scholar]

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[1] World health organization. World malaria report. Geneva : WHO, 2012. [Google Scholar]

[2] Miller LH, Baruch DI, Marsh K, Doumbo OK. The pathogenic basis of malaria. Nature 2002 ; 415 : 673–679. [CrossRef] [PubMed] [Google Scholar]

[3] Marti M, Good RT, Rug M, et al. Targeting malaria virulence and remodeling proteins to the host erythrocyte. Science 2004 ; 306 : 1930–1933. [CrossRef] [PubMed] [Google Scholar]

[4] Coban C, Ishii KJ, Kawai T, et al. Toll-like receptor 9 mediates innate immune activation by the malaria pigment hemozoin. J Exp Med 2005 ; 201 : 19–25. [CrossRef] [PubMed] [Google Scholar]

[5] Schofield L, Hewitt MC, Evans K, et al. Synthetic GPI as a candidate anti-toxic vaccine in a model of malaria. Nature 2002 ; 418 : 785–789. [CrossRef] [PubMed] [Google Scholar]

[6] Nantakomol D, Dondorp AM, Krudsood S, et al. Circulating red cell-derived microparticles in human malaria. J Infect Dis 2011 ; 203 : 700–706. [CrossRef] [PubMed] [Google Scholar]

[7] Pankoui Mfonkeu JB, Gouado I, Fotso Kuate H, et al. Elevated cell-specific microparticles are a biological marker for cerebral dysfunctions in human severe malaria. PLoS One 2010 ; 5 : e13415. [CrossRef] [PubMed] [Google Scholar]

[8] Couper KN, Barnes T, Hafalla JC, et al. Parasite-derived plasma microparticles contribute significantly to malaria infection-induced inflammation through potent macrophage stimulation. PLoS Pathog 2010 ; 6 : e1000744. [CrossRef] [PubMed] [Google Scholar]

[9] Mantel PY, Hoang AN, Goldowitz I, et al. Malaria-infected erythrocyte-derived microvesicles mediate cellular communication within the parasite population and with the host immune system. Cell Host Microbe 2013 ; 13 : 521–534. [CrossRef] [PubMed] [Google Scholar]

[10] Regev-Rudzki N, Wilson DW, Carvalho TG, et al. Cell-cell communication between malaria-infected red blood cells via exosome-like vesicles. Cell 2013 ; 153 : 1120–1133. [CrossRef] [PubMed] [Google Scholar]

[11] Baunaure F, Langsley G. Trafic protéique dans le globule rouge infecté par Plasmodium. Med Sci (Paris) 2005 ; 21 : 523–529. [CrossRef] [EDP Sciences] [PubMed] [Google Scholar]