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Accueil Tous les numéros Volume 23 / Numéro 11 (Novembre 2007) Med Sci (Paris), 23 11 (2007) 939-943 Références
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Numéro
Med Sci (Paris)
Volume 23, Numéro 11, Novembre 2007
Page(s) 939 - 943
Section M/S revues
DOI https://doi.org/10.1051/medsci/20072311939
Publié en ligne 15 novembre 2007
  1. Hardy P, Beauchamp M, Sennlaub F, et al. New insights into the retinal circulation : inflammatory lipid mediators in ischemic retinopathy. Prostaglandins Leukot Essent Fatty Acids 2005; 72 : 301–25. [Google Scholar]
  2. Gilbert C, Rahi J, Eckstein M, et al. Retinopathy of prematurity in middle-income countries. Lancet 1997; 350 : 12–4. [Google Scholar]
  3. Squadrito GL, Pryor WA. Oxidative chemistry of nitric oxide : the roles of superoxide, peroxynitrite, and carbon dioxide. Free Radic Biol Med 1998; 25 : 392–403. [Google Scholar]
  4. Kroncke KD. Mechanisms and biological consequences of nitrosative stress. Biol Chem 2003; 384 : 1341. [Google Scholar]
  5. Kermorvant-Duchemin E, Sennlaub F, Sirinyan M, et al. Trans-arachidonic acids generated during nitrative stress induce a thrombospondin-1-dependent microvascular degeneration. Nat Med 2005; 11 : 1339–45. [Google Scholar]
  6. Balazy M, Poff CD. Biological nitration of arachidonic acid. Curr Vasc Pharmacol 2004; 2 : 81–93. [Google Scholar]
  7. Holmes JM, Leske DA, Zhang S. The effect of raised inspired carbon dioxide on normal retinal vascular development in the neonatal rat. Curr Eye Res 1997; 16 : 78–81. [Google Scholar]
  8. Harris A, Arend O, Wolf S, et al. CO2 dependence of retinal arterial and capillary blood velocity. Acta Ophthalmol Scand 1995; 73 : 421–4. [Google Scholar]
  9. Radi R. Peroxynitrite reactions and diffusion in biology. Chem Res Toxicol 1998; 11 : 720–1. [Google Scholar]
  10. Johnson BA, Weil MH. Redefining ischemia due to circulatory failure as dual defects of oxygen deficits and of carbon dioxide excesses. Crit Care Med 1991; 19 : 1432–8. [Google Scholar]
  11. D’Arcangelo D, Facchiano F, Barlucchi LM, et al. Acidosis inhibits endothelial cell apoptosis and function and induces basic fibroblast growth factor and vascular endothelial growth factor expression. Circ Res 2000; 86 : 312–8. [Google Scholar]
  12. Checchin D, Sennlaub F, Sirinyan M, et al. Hypercapnia prevents neovascularization via nitrative stress. Free Radic Biol Med 2006; 40 : 543–53. [Google Scholar]
  13. Zhang ZG, Chopp M, Zaloga C, et al. Cerebral endothelial nitric oxide synthase expression after focal cerebral ischemia in rats. Stroke 1993; 24 : 2016–22. [Google Scholar]
  14. Depre C, Fierain L, Hue L. Activation of nitric oxide synthase by ischaemia in the perfused heart. Cardiovasc Res 1997; 33 : 82–7. [Google Scholar]
  15. Jiang H, Kruger N, Lahiri DR, et al. Nitrogen dioxide induces cis-trans-isomerization of arachidonic acid within cellular phospholipids. Detection of trans-arachidonic acids in vivo. J Biol Chem 1999; 274 : 16235–41. [Google Scholar]
  16. Zghibeh CM, Raj Gopal V, Poff CD, et al. Determination of trans-arachidonic acid isomers in human blood plasma. Anal Biochem 2004; 332 : 137–44. [Google Scholar]
  17. Beauchamp MH, Sennlaub F, Speranza G, et al. Redox-dependent effects of nitric oxide on microvascular integrity in oxygen-induced retinopathy. Free Radic Biol Med 2004; 37 : 1885–94. [Google Scholar]
  18. Dawson DW, Pearce SF, Zhong R, et al. CD36 mediates the in vitro inhibitory effects of thrombospondin-1 on endothelial cells. J Cell Biol 1997; 138 : 707–17. [Google Scholar]
  19. Nor JE, Mitra RS, Sutorik MM, et al. Thrombospondin-1 induces endothelial cell apoptosis and inhibits angiogenesis by activating the caspase death pathway. J Vasc Res 2000; 37 : 209–18. [Google Scholar]
  20. Wang S, Wu Z, Sorenson CM, et al. Thrombospondin-1-deficient mice exhibit increased vascular density during retinal vascular development and are less sensitive to hyperoxia-mediated vessel obliteration. Dev Dyn 2003; 228 : 630–42. [Google Scholar]
  21. Llorens S, Nava E. Cardiovascular diseases and the nitric oxide pathway. Curr Vasc Pharmacol 2003; 1 : 335–46. [Google Scholar]

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