Free Access
Issue
Med Sci (Paris)
Volume 26, Number 10, Octobre 2010
Page(s) 830 - 835
Section M/S revues
DOI https://doi.org/10.1051/medsci/20102610830
Published online 15 October 2010
  1. Danial NN, Korsmeyer SJ. Cell death: critical control points. Cell 2004 ; 116 : 205-19. [Google Scholar]
  2. Green DR, Kroemer G. The pathophysiology of mitochondrial cell death. Science 2004 ; 305 : 626-9. [Google Scholar]
  3. Desagher S, Martinou JC. Mitochondria as the central control point of apoptosis. Trends Cell Biol 2000 ; 10 : 369-77. [Google Scholar]
  4. Frank S, Gaume B, Bergmann-Leitner ES, et al. The role of dynamin-related protein 1, a mediator of mitochondrial fission, in apoptosis. Dev Cell 2001 ; 1 : 515-25. [Google Scholar]
  5. Youle RJ, Karbowski M. Mitochondrial fission in apoptosis. Nat Rev Mol Cell Biol 2005 ; 6 : 657-63. [Google Scholar]
  6. Lenaers G, Amati-Bonneau P, Delettre C, et al. De la levure aux maladies neurodégénératives : 10 ans d’exploration des pathologies de la dynamique mitochondriale. MedSci (Paris) 2010 ; 26 : 836-41. [Google Scholar]
  7. Sauvanet C, Arnauné-Pelloquin L, David C, et al. Dynamique et morphologie mitochondriales : acteurs, mécanismes et pertinence fonctionnelle. Med Sci (Paris) 2010 ; 26 : 823-9. [Google Scholar]
  8. Arnoult D. Mitochondrial fragmentation in apoptosis. Trends Cell Biol 2007 ; 17 : 6-12. [Google Scholar]
  9. Autret A, Martin SJ. Emerging role for members of the Bcl-2 family in mitochondrial morphogenesis. Mol Cell 2009 ; 36 : 355-63. [Google Scholar]
  10. Sheridan C, Delivani P, Cullen SP, Martin SJ. Bax- or Bak-induced mitochondrial fission can be uncoupled from cytochrome c release. Mol Cell 2008 ; 31 : 570-85. [Google Scholar]
  11. Arnoult D, Grodet A, Lee YJ, et al. Release of OPA1 during apoptosis participates in the rapid and complete release of cytochrome c and subsequent mitochondrial fragmentation. J Biol Chem 2005 ; 280 : 35742-50. [Google Scholar]
  12. Karbowski M, Lee YJ, Gaume B, et al. Spatial and temporal association of Bax with mitochondrial fission sites, Drp1, and Mfn2 during apoptosis. J Cell Biol 2002 ; 159 : 931-8. [Google Scholar]
  13. Karbowski M, Arnoult D, Chen H, et al. Quantitation of mitochondrial dynamics by photolabeling of individual organelles shows that mitochondrial fusion is blocked during the Bax activation phase of apoptosis. J Cell Biol 2004 ; 164 : 493-9. [Google Scholar]
  14. Brooks C, Wei Q, Feng L, et al. Bak regulates mitochondrial morphology and pathology during apoptosis by interacting with mitofusins. Proc Natl Acad Sci USA 2007 ; 104 : 11649-54. [Google Scholar]
  15. Breckenridge DG, Stojanovic M, Marcellus RC, Shore GC. Caspase cleavage product of BAP31 induces mitochondrial fission through endoplasmic reticulum calcium signals, enhancing cytochrome c release to the cytosol. J Cell Biol 2003 ; 160 : 1115-27. [Google Scholar]
  16. Germain M, Mathai JP, McBride HM, Shore GC. Endoplasmic reticulum BIK initiates DRP1-regulated remodelling of mitochondrial cristae during apoptosis. EMBOJ 2005 ; 24 : 1546-56. [Google Scholar]
  17. Ishihara N, Nomura M, Jofuku A, et al. Mitochondrial fission factor Drp1 is essential for embryonic development and synapse formation in mice. Nat Cell Biol 2009 ; 11 : 958-66. [Google Scholar]
  18. Estaquier J, Arnoult D. Inhibiting Drp1-mediated mitochondrial fission selectively prevents the release of cytochrome c during apoptosis. Cell Death Differ 2007 ; 14 : 1086-94. [Google Scholar]
  19. Parone PA, James DI, Da Cruz S, et al. Inhibiting the mitochondrial fission machinery does not prevent Bax/Bak-dependent apoptosis. Mol Cell Biol 2006 ; 26 : 7397-408. [Google Scholar]
  20. Scorrano L, Ashiya M, Buttle K, et al. A distinct pathway remodels mitochondrial cristae and mobilizes cytochrome c during apoptosis. Dev Cell 2002 ; 2 : 55-67. [Google Scholar]
  21. Frezza C, Cipolat S, Martins de Brito O, et al. OPA1 controls apoptotic cristae remodeling independently from mitochondrial fusion. Cell 2006 ; 126 : 177-89. [Google Scholar]
  22. Yamaguchi R, Lartigue L, Perkins G, et al. Opa1-mediated cristae opening is Bax/Bak and BH3 dependent, required for apoptosis, and independent of Bak oligomerization. Mol Cell 2008 ; 31 : 557-69. [Google Scholar]
  23. Berman SB, Chen YB, Qi B, et al. Bcl-xL increases mitochondrial fission, fusion, and biomass in neurons. J Cell Biol 2009 ; 184 : 707-19. [Google Scholar]
  24. Karbowski M, Norris KL, Cleland MM, et al. Role of Bax and Bak in mitochondrial morphogenesis. Nature 2006 ; 443 : 658-62. [Google Scholar]
  25. Delivani P, Adrain C, Taylor RC, et al. Role for CED-9 and Egl-1 as regulators of mitochondrial fission and fusion dynamics. Mol Cell 2006 ; 21 : 761-73. [Google Scholar]
  26. Arnoult D, Rismanchi N, Grodet A, et al. Bax/Bak-dependent release of DDP/TIMM8a promotes Drp1-mediated mitochondrial fission and mitoptosis during programmed cell death. Curr Biol 2005 ; 15 : 2112-8. [Google Scholar]
  27. Xue L, Fletcher GC, Tolkovsky AM. Mitochondria are selectively eliminated from eukaryotic cells after blockade of caspases during apoptosis. Curr Biol 2001 ; 11 : 361-5. [Google Scholar]
  28. Barsoum MJ, Yuan H, Gerencser AA, et al. Nitric oxide-induced mitochondrial fission is regulated by dynamin-related GTPases in neurons. EMBOJ 2006 ; 25 : 3900-11. [Google Scholar]
  29. Kroemer G, Levine B. Autophagic cell death: the story of a misnomer. Nat Rev Mol Cell Biol 2008 ; 9 : 1004-10. [Google Scholar]

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