Free Access
Issue
Med Sci (Paris)
Volume 23, Number 5, Mai 2007
Page(s) 497 - 501
Section M/S revues
DOI https://doi.org/10.1051/medsci/2007235497
Published online 15 May 2007
  1. Annesi-Maesano I, Dab W. Air pollution and the lung: epidemiological approach. Med Sci (Paris) 2006; 22 : 589–94. [Google Scholar]
  2. Xiao GG, Wang M, Li N, et al. Use of proteomics to demonstrate a hierarchical oxidative stress response to Diesel exhaust particle chemicals in a macrophage cell line. J Biol Chem 2003; 278 : 50781–90. [Google Scholar]
  3. Li N, Hao M, Phalen RF, et al. Particulate air pollutants and asthma. A paradigm for the role of oxidative stress in PM-induced adverse health effects. Clin Immunol 2003; 109 : 250–65. [Google Scholar]
  4. MacNee W, Donaldson K. Mechanisms of lung injury caused by PM10 and ultrafine particles with special reference to COPD. Eur Respir J 2003; 21 : 47–51. [Google Scholar]
  5. Wilson MR, Lightbody JH, Donaldson K, et al. Interactions between ultrafine particles and transition metals in vivo and in vitro. Toxicol Appl Pharmacol 2002; 184 : 172–9. [Google Scholar]
  6. Donaldson K, Brown DM, Mitchell C, et al. Free radical activity of PM10: iron-mediated generation of hydroxyl radicals. Environ Health Perspect 1997; 105 : 1285–9. [Google Scholar]
  7. Li N, Sioutas C, Cho A, et al. Ultrafine particulate pollutants induce oxidative stress and mitochondrial damage. Environ Health Perspect 2003; 111 : 455–60. [Google Scholar]
  8. Hiura TS, Kaszubowski MP, Li N, Nel AE. Chemicals in diesel exhaust particles generate reactive oxygen radicals and induce apoptosis in macrophages. J Immunol 1999; 163 : 5582–91. [Google Scholar]
  9. Baulig A, Garlatti M, Bonvallot V, et al. Involvement of reactive oxygen species in the metabolic pathways triggered by diesel exhaust particles in human airway epithelial cells. Am J Physiol Lung Cell Mol Physiol 2003; 285 : L671–9. [Google Scholar]
  10. Squadrito GL, Cueto R, Dellinger B, Pryor W. Quinoid redox cycling as a mechanism for sustained free radical generation by inhaled airborne particulate matter. Free Rad Biol Med 2001; 31 : 1132–8. [Google Scholar]
  11. Krishna MT, Chauhan AJ, Frew AJ, Holgate ST. Toxicological mechanisms underlying oxidant pollutant-induced airway injury. Rev Environ Health 1998; 13 : 59–71. [Google Scholar]
  12. Nichols BG, Woods JS, Luchtel DL, et al. Effects of ozone exposure on nuclear factor-kappaB activation and tumor necrosis factor-alpha expression in human nasal epithelial cells. Toxicol Sci 2001; 60 : 356–62. [Google Scholar]
  13. Gilmour PS, Ziesenis A, Morrison ER, et al. Pulmonary and systemic effects of short-term inhalation exposure to ultrafine carbon black particles. Toxicol Appl Pharmacol 2004; 195 : 35–44. [Google Scholar]
  14. Ghio AJ, Devlin RB. Inflammatory lung injury after bronchial instillation of air pollution particles. Am J Respir Crit Care Med 2001; 164 : 704–8. [Google Scholar]
  15. Bonvallot V, Baeza-Squiban A, Baulig A, et al. Organic compounds from Diesel exhaust particles elicit a proinflammatory response in human airway epithelial cells and induce cytochrome p450 1A1 expression. Am J Respir Cell Mol Biol 2001; 25 : 515–21. [Google Scholar]
  16. Li N, Nel E. Role of the Nrf2-mediated signalling pathway as a negative regulator of inflammation: implications for the impact of particulate pollutants on asthma. Antiox Redox Signal 2006; 8 : 88–98. [Google Scholar]
  17. Nemmar A, Hoet PH, Vanquickenborne B, et al. Passage of inhaled particles into the blood circulation in humans. Circulation 2002; 105 : 411–4. [Google Scholar]
  18. Churg A, Brauer M, del Carmen Avila-Casado M, et al. Chronic exposure to high levels of particulate air pollution and small airway remodeling. Environ Health Perspect 2003; 111 : 714–8. [Google Scholar]
  19. Hiura TS, Li N, Kaplan R, et al.The role of a mitochondrial pathway in the induction of apoptosis by chemicals extracted from Diesel exhaust particles. J Immunol 2000; 165 : 2703–11. [Google Scholar]
  20. Bayram H, Ito K, Issa R, et al. Regulation of human lung epithelial cell numbers by diesel exhaust particles. Eur Respir J 2006; 27 : 705–13. [Google Scholar]
  21. Bonner JC. The epidermal growth factor receptor at the crossroads of airway remodeling. Am J Physiol Lung Cell Mol Physiol 2002; 283 : L528–30. [Google Scholar]
  22. Wu W, Samet JM, Ghio AJ, Devlin RB. Activation of the EGF receptor signalling pathway in airway epithelial cells exposed to Utah valley PM. Am J Physiol Lung Cell Mol Physiol 2001; 281 : L483–9. [Google Scholar]
  23. Carpenter G. Employment of the epidermal growth factor receptor in growth factor-independent signaling pathways. J Cell Biol 1999; 146 : 697–702. [Google Scholar]
  24. Blanchet S, Ramgolam K, Baulig A, Marano F, Baeza-Squiban A. Fine particulate matter induces amphiregulin secretion by bronchial epithelial cells. Am J Respir Cell Mol Biol 2004; 30 : 421–7. [Google Scholar]
  25. Lemjabbar H, Li D, Gallup M, et al. Tobacco smoke-induced lung cell proliferation mediated by tumor necrosis factor (alpha)-converting enzyme and amphiregulin. J Biol Chem 2003; 278: 26202–7. [Google Scholar]
  26. Auger F, Gendron MC, Chamot C, et al. Responses of well-differentiated nasal epithelial cells exposed to particles: role of the epithelium in airway inflammation. Toxicol Appl Pharmacol 2006; 215 : 285–9 [Google Scholar]

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