Issue |
Med Sci (Paris)
Volume 24, Number 2, Février 2008
|
|
---|---|---|
Page(s) | 139 - 141 | |
Section | Nouvelles | |
DOI | https://doi.org/10.1051/medsci/2008242139 | |
Published online | 15 February 2008 |
GDF15 coupable de l’hypersidérémie des patients thalassémiques par extinction de l’hepcidine
Erythroblasts-derived GDF15 supresses hepcidin in thalassemia
Institut Cochin, Université Paris Descartes, CNRS (UMR 8104), Paris, France. Inserm, U567, Paris, France.
Département de génétique, développement et pathologie moléculaire, Institut Cochin, 24, rue du Faubourg Saint-Jacques, 75014 Paris, France
*
vaulont@cochin.inserm.fr
*
labie@cochin.inserm.fr
Cet article ne possède pas de résumé.
© 2008 médecine/sciences - Inserm / SRMS
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