Accès gratuit
Numéro
Med Sci (Paris)
Volume 32, Numéro 1, Janvier 2016
Origine développementale de la santé et des maladies (DOHaD), environnement et épigénétique
Page(s) 51 - 56
Section M/S Revues
DOI https://doi.org/10.1051/medsci/20163201009
Publié en ligne 5 février 2016
  1. WHO/UNEP. State of the science of endocrine disrupting chemicals – 2012. An assessment of the state of the science of endocrine disruptors prepared by a group of experts for the United Nations Environment Programme (UNEP) and WHO. Geneva : WHO, 2013 : 296 p.
  2. Casals-Casas C, Desvergne B. Endocrine disruptors: from endocrine to metabolic disruption. Ann Rev Physiol 2011 ; 73 : 135–162. [CrossRef]
  3. Thayer KA, Heindel JJ, Bucher JR, Gallo MA. Role of environmental chemicals in diabetes and obesity: a National toxicology program workshop review. Environ Health Perspect 2012 ; 120 : 779–789. [CrossRef] [PubMed]
  4. Ruzzin J, Petersen R, Meugnier E, et al. Persistent organic pollutant exposure leads to insulin resistance syndrome. Environ Health Perspect 2010 ; 118 : 465–471. [CrossRef] [PubMed]
  5. Le Magueresse-Battistoni B, Vidal H, Naville D. Lifelong consumption of low-dosed food pollutants and metabolic health. J Epidemiol Community Health 2015 ; 69 : 512–515. [CrossRef] [PubMed]
  6. WHO. Obesity and Overweight. WHO, 2014: Fact sheet 311. http://www.whoint/mediacentre/factsheets/fs311/en/.
  7. De Onis M, Blossner M, Borghi E. Global prevalence and trends of overweight and obesity among preschool children. Am J Clin Nutr 2010 ; 9 2: 1257–1264. [CrossRef]
  8. Legler J, Fletcher T, Govarts E, et al. Obesity, diabetes, and associated costs of exposure to endocrine-disrupting chemicals in the European Union. J Clin Endocrinol Metab 2015 ; 100 : 1278–1288. [CrossRef] [PubMed]
  9. Cravedi JP, Zalko D, Savouret JF, et al. Le concept de perturbation endocrinienne et la santé humaine. Med Sci (Paris) 2007 ; 23 : 198–204. [CrossRef] [EDP Sciences] [PubMed]
  10. Mauvais-Jarvis F, Clegg DJ, Hevener AL. The role of estrogens in control of energy balance and glucose homeostasis. Endocr Rev 2013 ; 34 : 309–338. [CrossRef] [PubMed]
  11. Vandenberg LN, Colborn T, Hayes TB, et al. Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses. Endocr Rev 2012 ; 33 : 378–455. [CrossRef] [PubMed]
  12. Barouki R, Gluckman PD, Grandjean P, et al. Developmental origins of non-communicable disease: implications for research and public health. Environ Health 2012 ; 11 : 42. [CrossRef] [PubMed]
  13. Barker DJ, Eriksson JG, Forsen T, Osmond C. Fetal origins of adult disease: strength of effects and biological basis. Int J Epidemiol 2002 ; 31 : 1235–1239. [CrossRef] [PubMed]
  14. Lee DH, Lee IK, Song K, et al. A strong dose-response relation between serum concentrations of persistent organic pollutants and diabetes: results from the National Health and Examination Survey 1999–2002. Diabetes Care 2006 ; 29 : 1638–1644. [CrossRef] [PubMed]
  15. Milbrath MO, Wenger Y, Chang CW, et al. Apparent half-lives of dioxins, furans, and polychlorinated biphenyls as a function of age, body fat, smoking status, and breast-feeding. Environ Health Perspect 2009 ; 117 : 417–425. [CrossRef] [PubMed]
  16. Grun F, Blumberg B. Perturbed nuclear receptor signaling by environmental obesogens as emerging factors in the obesity crisis. Rev Endocr Metab Disord 2007 ; 8 : 161–171. [CrossRef] [PubMed]
  17. Baillie-Hamilton PF. Chemical toxins: a hypothesis to explain the global obesity epidemic. J Altern Complement Med 2002 ; 8 : 185–192. [CrossRef]
  18. Kirchner S, Kieu T, Chow C, et al. Prenatal exposure to the environmental obesogen tributyltin predisposes multipotent stem cells to become adipocytes. Mol Endocrinol 2010 ; 24 : 526–539. [CrossRef]
  19. Chamorro-Garcia R, Sahu M, Abbey RJ, et al. Transgenerational inheritance of increased fat depot size, stem cell reprogramming, and hepatic steatosis elicited by prenatal exposure to the obesogen tributyltin in mice. Environ Health Perspect 2013 ; 121 : 359–366. [CrossRef] [PubMed]
  20. Skinner MK. Endocrine disruptor induction of epigenetic transgenerational inheritance of disease. Mol Cell Endocrinol 2014 ; 398 : 4–12. [CrossRef] [PubMed]
  21. Bastos Sales L, Kamstra JH, Cenijn PH, et al. Effects of endocrine disrupting chemicals on in vitro global DNA methylation and adipocyte differentiation. Toxicol In Vitro 2013; 27 : 1634–1643. [CrossRef] [PubMed]
  22. Newbold RR, Padilla-Banks E, Snyder RJ, Jefferson WN. Developmental exposure to estrogenic compounds and obesity. Birth Defects Res A Clin Mol Teratol 2005 ; 73 : 478–480. [CrossRef] [PubMed]
  23. Verhulst SL, Nelen V, Hond ED, et al. Intrauterine exposure to environmental pollutants and body mass index during the first 3 years of life. Environ Health Perspect 2009 ; 117 : 122–126. [CrossRef] [PubMed]
  24. Dang ZC, Lowik C. Dose-dependent effects of phytoestrogens on bone. Trends Endocrinol Metab 2005 ; 16 : 207–213. [CrossRef] [PubMed]
  25. Yoon M. PPARalpha in obesity: Sex difference and estrogen involvement. PPAR Res 2010 ; 2010 : 584296. [CrossRef] [PubMed]
  26. Tohme M, Prud’homme SM, Boulahtouf A, et al. Estrogen-related receptor gamma is an in vivo receptor of bisphenol A. FASEB J 2014 ; 28 : 3124–3133. [CrossRef] [PubMed]
  27. Frederiksen H, Nielsen JK, Morck TA, et al. Urinary excretion of phthalate metabolites, phenols and parabens in rural and urban Danish mother-child pairs. Int J Hyg Environ Health 2013 ; 216 : 772–783. [CrossRef] [PubMed]
  28. Rantakokko P, Main KM, Wohlfart-Veje C, et al. Association of placenta organotin concentrations with growth and ponderal index in 110 newborn boys from Finland during the first 18 months of life: a cohort study. Environ Health 2014 ; 13 : 45. [CrossRef] [PubMed]
  29. De Cock M, van de Bor M. Obesogenic effects of endocrine disruptors, what do we know from animal and human studies? Environ Int 2014 ; 70 : 15–24. [CrossRef]
  30. Valvi D, Mendez MA, Martinez D, et al. Prenatal concentrations of polychlorinated biphenyls, DDE, and DDT and overweight in children: a prospective birth cohort study. Environ Health Perspect 2012 ; 120 : 451–457. [CrossRef] [PubMed]
  31. Warner M, Wesselink A, Harley KG, et al. Prenatal exposure to dichlorodiphenyltrichloroethane and obesity at 9 years of age in the Chamacos study cohort. Am J Epidemiol 2014 ; 179 : 1312–1322. [CrossRef] [PubMed]
  32. Naville D, Pinteur C, Vega N, et al. Low-dose food contaminants trigger sex-specific, hepatic metabolic changes in the progeny of obese mice. FASEB J 2013 ; 27 : 3860–3870. [CrossRef] [PubMed]
  33. Mauduit C, Siddeek B, Benahmed M. Origine développementale et environnementale de l’infertilité : rôle des perturbateurs hormonaux. Med Sci (Paris) 2016 ; 32 : 45–50. [CrossRef] [EDP Sciences] [PubMed]

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