Hyperprolactinémie et infertilité - Approche physiopathologique

Charlotte Sonigo; Jacques Young; Nadine Binart

doi:10.1051/medsci/2013293004

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Volume 29 / Numéro 3 (Mars 2013)

Med Sci (Paris), 29 3 (2013) 242-244

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Numéro		Med Sci (Paris) Volume 29, Numéro 3, Mars 2013


Page(s)		242 - 244
Section		Nouvelles
DOI		https://doi.org/10.1051/medsci/2013293004
Publié en ligne		27 mars 2013

Sonigo C, Bouilly J, Carré N, et al. Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration. J Clin Invest 2012 ; 122 : 3791–3795. [CrossRef] [PubMed] [Google Scholar]
Bouchard P, Lagoguey M, Brailly S, Schaison G. Gonadotropin-releasing hormone pulsatile administration restores luteinizing hormone pulsatility and normal testosterone levels in males with hyperprolactinemia. J Clin Endocrinol Metab 1985 ; 60 : 258–262. [CrossRef] [PubMed] [Google Scholar]
Milenković L, D’Angelo G, Kelly PA, Weiner RI. Inhibition of gonadotropin hormone-releasing hormone release by prolactin from GT1 neuronal cell lines through prolactin receptors. Proc Natl Acad Sci USA 1994 ; 91 : 1244–1247. [CrossRef] [Google Scholar]
Grattan DR, Jasoni CL, Liu X, et al. Prolactin regulation of gonadotropin-releasing hormone neurons to suppress luteinizing hormone secretion in mice. Endocrinology 2007 ; 148 : 4344–4351. [CrossRef] [PubMed] [Google Scholar]
de Roux N, Genin E, Carel J-C, et al. Hypogonadotropic hypogonadism due to loss of function of the KiSS1-derived peptide receptor GPR54. Proc Natl Acad Sci USA 2003 ; 100 : 10972–10976. [CrossRef] [Google Scholar]
Seminara SB, Messager S, Chatzidaki EE, et al. The GPR54 gene as a regulator of puberty. N Engl J Med 2003 ; 349 : 1614–1627. [CrossRef] [PubMed] [Google Scholar]
Roa J, Navarro VM, Tena-Sempere M. Kisspeptins in reproductive biology: consensus knowledge and recent developments. Biol Reprod 2011 ; http://www.ncbi.nlm.nih.gov/pubmed/21677307 [Google Scholar]
Kokay IC, Petersen SL, Grattan DR. Identification of prolactin-sensitive GABA and kisspeptin neurons in regions of the rat hypothalamus involved in the control of fertility. Endocrinology 2011 ; 152 : 526–535. [CrossRef] [PubMed] [Google Scholar]

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[1] Sonigo C, Bouilly J, Carré N, et al. Hyperprolactinemia-induced ovarian acyclicity is reversed by kisspeptin administration. J Clin Invest 2012 ; 122 : 3791–3795. [CrossRef] [PubMed] [Google Scholar]

[2] Bouchard P, Lagoguey M, Brailly S, Schaison G. Gonadotropin-releasing hormone pulsatile administration restores luteinizing hormone pulsatility and normal testosterone levels in males with hyperprolactinemia. J Clin Endocrinol Metab 1985 ; 60 : 258–262. [CrossRef] [PubMed] [Google Scholar]

[3] Milenković L, D’Angelo G, Kelly PA, Weiner RI. Inhibition of gonadotropin hormone-releasing hormone release by prolactin from GT1 neuronal cell lines through prolactin receptors. Proc Natl Acad Sci USA 1994 ; 91 : 1244–1247. [CrossRef] [Google Scholar]

[4] Grattan DR, Jasoni CL, Liu X, et al. Prolactin regulation of gonadotropin-releasing hormone neurons to suppress luteinizing hormone secretion in mice. Endocrinology 2007 ; 148 : 4344–4351. [CrossRef] [PubMed] [Google Scholar]

[5] de Roux N, Genin E, Carel J-C, et al. Hypogonadotropic hypogonadism due to loss of function of the KiSS1-derived peptide receptor GPR54. Proc Natl Acad Sci USA 2003 ; 100 : 10972–10976. [CrossRef] [Google Scholar]

[6] Seminara SB, Messager S, Chatzidaki EE, et al. The GPR54 gene as a regulator of puberty. N Engl J Med 2003 ; 349 : 1614–1627. [CrossRef] [PubMed] [Google Scholar]

[7] Roa J, Navarro VM, Tena-Sempere M. Kisspeptins in reproductive biology: consensus knowledge and recent developments. Biol Reprod 2011 ; http://www.ncbi.nlm.nih.gov/pubmed/21677307 [Google Scholar]

[8] Kokay IC, Petersen SL, Grattan DR. Identification of prolactin-sensitive GABA and kisspeptin neurons in regions of the rat hypothalamus involved in the control of fertility. Endocrinology 2011 ; 152 : 526–535. [CrossRef] [PubMed] [Google Scholar]