Authentification abonné
medsci
Accueil Tous les numéros Volume 27 / Numéro 12 (Décembre 2011) Med Sci (Paris), 27 12 (2011) 1089-1095 Références
Accès gratuit
Numéro
Med Sci (Paris)
Volume 27, Numéro 12, Décembre 2011
Page(s) 1089 - 1095
Section M/S Revues
DOI https://doi.org/10.1051/medsci/20112712015
Publié en ligne 23 décembre 2011
  1. Zeller KI, Jegga AG, Aronow BJ, et al. An integrated database of genes responsive to the Myc oncogenic transcription factor: identification of direct genomic targets. Genome Biol 2003 ; 4 : R69. [CrossRef] [PubMed]
  2. Da Cruz S, Xenarios I, Langridge J, et al. Proteomic analysis of the mouse liver mitochondrial inner membrane. J Biol Chem 2003 ; 278 : 41566–41571. [CrossRef] [PubMed]
  3. Frickey T, Lupas AN. Phylogenitic analysis of AAA proteins. J Struct Biol 2004 ; 146 : 2–10. [CrossRef] [PubMed]
  4. Kamath RS, Ahringer J. Genome-wide RNAi screening in Caenorhabditis elegans. Methods 2003 ; 30 : 313–321. [CrossRef] [PubMed]
  5. Hoffmann M, Bellance N, Rossignol R, et al. C. elegans ATAD-3 is essential for mitochondrial activity and development. PLoS One 2009 ; 4 : e7644. [CrossRef] [PubMed]
  6. Gilquin B, Taillebourg E, Cherradi N, et al. The AAA+ ATPase ATAD3A controls mitochondrial dynamics at the interface of the inner and outer membrane. Mol Cell Biol 2010 ; 30 : 1984–1996. [CrossRef] [PubMed]
  7. Hubstenberger A. MSBP, une protéine identifiée comme une cible de la S100B, impliquée dans la distribution subcellulaire des mitochondries. Grenoble, Thèse de médecine, Université Joseph Fourier, 2006. http://tel..archives-ouvertes.fr/docs/00/17/52/59/PDF/Arnaud_Hubstenberger.pdf.
  8. Hubstenberger A, Labourdette G, Baudier J, Rousseau D. ATAD3A and ATAD3B are distal 1p-located genes differentially expressed in human glioma cell lines and present in vitro anti-oncogenic and chemoresistant properties. Exp Cell Res 2008 ; 314 : 2870–2883. [CrossRef] [PubMed]
  9. Schaffrik M, Mack B, Matthias C, et al. Molecular characterization of the tumor-associated antigen AAA-TOB3. Cell Mol Life Sci 2006 ; 63 : 2162–2174. [CrossRef] [PubMed]
  10. He J, Mao CC, Reyes A, et al. The AAA+ protein ATAD3 has displacement loop binding properties and is involved in mitochondrial nucleoid organization. J Cell Biol 2007 ; 176 : 141–146. [CrossRef] [PubMed]
  11. Wang Y, Bogenhagen DF. Human mitochondrial DNA nucleoids are linked to protein folding machinery and metabolic enzymes at the mitochondrial inner membrane. J Biol Chem 2006 ; 281 : 25791–25802. [CrossRef] [PubMed]
  12. Bogenhagen DF, Rousseau D, Burke S. The layered structure of human mitochondrial DNA nucleoids. J Biol Chem 2008 ; 283 : 3665–3667. [CrossRef] [PubMed]
  13. Hubstenberger A, Merle N, Charton R, et al. Topological analysis of ATAD3A insertion in purified human mitochondria. J Bioenerg Biomembr 2010 ; 42 : 143–150. [CrossRef] [PubMed]
  14. Simmons DM, Voss JW, Ingraham HA, et al. Pituitary cell phenotypes involve cell-specific Pit-1 mRNA translation and synergistic interactions with other classes of transcription factors. Genes Dev 1990 ; 4 : 695–711. [NASA ADS] [CrossRef] [MathSciNet] [PubMed]
  15. Sassoon D, Lyons G, Wright WE, et al. Expression of two myogenic regulatory factors myogenin and MyoD1 during mouse embryogenesis. Nature 1989 ; 341 : 303–307. [CrossRef] [PubMed]
  16. Hinterberger TJ, Sassoon DA, Rhodes SJ, Konieczny SF. Expression of the muscle regulatory factor MRF4 during somite and skeletal myofiber development. Dev Biol 1991 ; 147 : 144–156. [CrossRef] [PubMed]
  17. Pownall ME, Gustafsson MK, Emerson CP Jr. Myogenic regulatory factors and the specification of muscle progenitors in vertebrate embryons. Annu Rev Cell Dev Biol 2002 ; 18 : 747–783. [CrossRef] [PubMed]
  18. Fang HY, Chang CL, Hsu SH, et al. ATPase family AAA domain-containing 3A is a novel anti-apoptotic factor in lung adenocarcinoma cells. J Cell Sci 2010 ; 123 : 1171–1180. [CrossRef] [PubMed]
  19. Hayashi T, Rizzuto R, Hajnoczky G, Su TP. MAM: more than just a housekeeper. Trends Cell Biol 2009 ; 19 : 81–88. [CrossRef] [PubMed]
  20. Smith JB, Alderete B, Minn Y, et al. Localization of common deletion regions on 1p and 19q in human glioma and their association with histological subtype. Oncogene 1999 ; 18 : 4144–4152. [CrossRef] [PubMed]
  21. Jiang Y, Liu X, Fang X, Wang X. Proteomic analysis of mitochondria in Raji cells following exposure to radiation: implications for radiotherapy response. Protein Pept Lett 2009 ; 16 : 1350–1359. [CrossRef] [PubMed]
  22. Gires O, Münz M, Schaffrik M, et al. Profile identification of disease-asociated humoral antigens using Amida, a novel proteomics-based technology. Cell Mol Life Sci 2004 ; 61 : 1198–1207. [CrossRef] [PubMed]
  23. Geuijen CA, Bijl N, Smit RC, et al. A proteomic approach to tumour target identification using phage display, affinity purification and mass spectrometry. Eur J Cancer 2005 ; 41 : 178–187. [CrossRef] [PubMed]
  24. Correia AR, Pastore C, Adinolfi S, et al. Dynamics, stability and iron-binding activity of frataxin clinical mutants. FEBS J 2008 ; 275 : 3680–3690. [CrossRef] [PubMed]
  25. Gilquin B, Cannon BR, Hubstenberger A, et al. The calcium-dependent interaction between S100B and the mitochondrial AAA-ATPase ATAD3A and the role of this complex in the cytoplasmic processing of ATAD3A. Mol Cell Biol 2010 ; 30 : 2724–2736. [CrossRef] [PubMed]
  26. Kornmann B. Le complexe Ermes : une connexion haut-débit entre le réticulum endoplasmique et les mitochondries. Med Sci (Paris) 2010 ; 26 : 145–146. [CrossRef] [EDP Sciences] [PubMed]
  27. Kornmann B, Walter P. ERMES-mediated ER-mitochondria contacts: molecular hubs for the regulation of mitochondrial biology. J Cell Sci 2010 ; 123 : 1389–1393. [CrossRef] [PubMed]

Les statistiques affichées correspondent au cumul d'une part des vues des résumés de l'article et d'autre part des vues et téléchargements de l'article plein-texte (PDF, Full-HTML, ePub... selon les formats disponibles) sur la platefome Vision4Press.

Les statistiques sont disponibles avec un délai de 48 à 96 heures et sont mises à jour quotidiennement en semaine.

Le chargement des statistiques peut être long.