Accès gratuit
Numéro
Med Sci (Paris)
Volume 24, Numéro 5, Mai 2008
Page(s) 464 - 466
Section Nouvelles
DOI https://doi.org/10.1051/medsci/2008245464
Publié en ligne 15 mai 2008
  1. Snider WD, Wright DE. Neurotrophins cause a new sensation. Neuron 1996; 16 : 229–32.
  2. Shatzky S, Moses S, Levy J, et al. Congenital insensitivity to pain with anhidrosis (CIPA) in Israeli-Bedouins : genetic heterogeneity, novel mutations in the TRKA/NGF receptor gene, clinical findings, and results of nerve conduction studies. Am J Med Genet 2000; 92 : 353–60.
  3. Molliver DC, Wright DE, Leitner ML, et al. IB4-binding DRG neurons switch from NGF to GDNF dependence in early postnatal life. Neuron 1997; 19 : 849–61.
  4. Malin SA, Molliver DC, Koerber HR, et al. Glial cell line-derived neurotrophic factor family members sensitize nociceptors in vitro and produce thermal hyperalgesia in vivo. J Neurosci 2006; 26 : 8588–99.
  5. Shu X, Mendell LM. Nerve growth factor acutely sensitizes the response of adult rat sensory neurons to capsaicin. Neurosci Lett 1999; 274 : 159–62.
  6. Chuang HH, Prescott ED, Kong H, et al. Bradykinin and nerve growth factor release the capsaicin receptor from PtdIns(4,5)P2-mediated inhibition. Nature 2001; 411 : 957–62.
  7. Caterina MJ, Schumacher MA, Tominaga M, et al. The capsaicin receptor : a heat-activated ion channel in the pain pathway. Nature 1997; 389 : 816–24.
  8. Caterina MJ, Leffler A, Malmberg AB, et al. Impaired nociception and pain sensation in mice lacking the capsaicin receptor. Science 2000; 288 : 306–13.
  9. Davis JB, Gray J, Gunthorpe MJ, et al. Vanilloid receptor-1 is essential for inflammatory thermal hyperalgesia. Nature 2000; 405 : 183–7.
  10. Szallasi A, Cortright DN, Blum CA, Eid SR. The vanilloid receptor TRPV1 : 10 years from channel cloning to antagonist proof-of-concept. Nat Rev Drug Discov 2007; 6 : 357–72.
  11. Vassort G, Fauconnier J. Les canaux TRP (transient receptor potential) : une nouvelle famille de canaux à expression variée. Med Sci (Paris) 2008; 24 : 163–8.
  12. Calvino B. L’hypersensibilité à la chaleur au cours de l’inflammation : le rôle du récepteur TRPV1. Med Sci (Paris) 2003; 19 : 649–51.
  13. Zhuang ZY, Xu H, Clapham DE, Ji RR. Phosphatidylinositol 3-kinase activates ERK in primary sensory neurons and mediates inflammatory heat hyperalgesia through TRPV1 sensitization. J Neurosci 2004; 24 : 8300–9.
  14. Zhu W, Oxford GS. Phosphoinositide-3-kinase and mitogen activated protein kinase signaling pathways mediate acute NGF sensitization of TRPV1. Mol Cell Neurosci 2007; 34 : 689–700.
  15. Zhang X, Huang J, McNaughton PA. NGF rapidly increases membrane expression of TRPV1 heat-gated ion channels. EMBO J 2005; 24 : 4211–23.
  16. Bonnington JK, McNaughton PA. Signalling pathways involved in the sensitisation of mouse nociceptive neurones by nerve growth factor. J Physiol 2003; 551 : 433–46.
  17. Hirata T, Morii E, Morimoto M, et al. Stem cell factor induces outgrowth of c-kit-positive neurites and supports the survival of c-kit-positive neurons in dorsal root ganglia of mouse embryos. Development 1993; 119 : 49–56.
  18. Milenkovic N, Frahm C, Gassmann M, et al. Nociceptive tuning by stem cell factor/c-Kit signaling. Neuron 2007; 56 : 893–906.
  19. Mandadi S, Tominaga T, Numazaki M, et al. Increased sensitivity of desensitized TRPV1 by PMA occurs through PKC-epsilon-mediated phosphorylation at S800. Pain 2006; 123 : 106–16.
  20. Shu X, Mendell LM. Acute sensitization by NGF of the response of small-diameter sensory neurons to capsaicin. J Neurophysiol 2001; 86 : 2931–8.

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