Accès gratuit
Med Sci (Paris)
Volume 22, Numéro 8-9, Août–Septembre 2006
Page(s) 739 - 744
Section M/S revues
Publié en ligne 15 août 2006
  1. Snow RW, Guerra CA, Noor AM, et al. The global distribution of clinical episodes of Plasmodium falciparum malaria. Nature 2005; 434 : 214–7
  2. Kwiatkowski D, Nowak M. Periodic and chaotic host-parasite interactions in human malaria. Proc Natl Acad Sci USA 1991; 88 : 5111–3.
  3. Marti M, Good RT, Rug M, et al. Targeting malaria virulence and remodeling proteins to the host erythrocyte. Science 2004; 306 : 1930–3.
  4. Schofield L, Grau GE. Immunological processes in malaria pathogenesis. Nat Rev Immunol 2005; 5 : 722–35.
  5. Bruce MC, Day KP. Cross-species regulation of Plasmodium parasitemia in semi-immune children from Papua New Guinea. Trends Parasitol 2003; 19 : 271–7.
  6. Favre N, Ryffel B, Bordmann G, et al. The course of Plasmodium chabaudi chabaudi infections in interferon-gamma receptor deficient mice. Parasite Immunol 1997; 19 : 375–83.
  7. Stevenson MM, Tam MF, Wolf SF, et al. IL-12-induced protection against blood-stage Plasmodium chabaudi AS requires IFN-gamma and TNF-alpha and occurs via a nitric oxide-dependent mechanism. J Immunol 1995; 155 : 2545–56.
  8. De Souza JB, Williamson KH, Otani T, et al. Early gamma interferon responses in lethal and nonlethal murine blood-stage malaria. Infect Immun 1997; 65 : 1593–8.
  9. Jacobs P, Radzioch D, Stevenson MM. A Th1-associated increase in tumor necrosis factor alpha expression in the spleen correlates with resistance to blood-stage malaria in mice. Infect Immun 1996; 64 : 535–41.
  10. Luty AJ, Lell B, Schmidt-Ott R, et al. Interferon-gamma responses are associated with resistance to reinfection with Plasmodium falciparum in young African children. J Infect Dis 1999; 179 : 980–8.
  11. Dodoo D, Omer FM, Todd J, et al. Absolute levels and ratios of proinflammatory and anti-inflammatory cytokine production in vitro predict clinical immunity to Plasmodium falciparum malaria. J Infect Dis 2002; 185 : 971–9.
  12. Kojima S, Nagamine Y, Hayano M, et al. A potential role of interleukin 18 in severe falciparum malaria. Acta Trop 2004; 89 : 279–84.
  13. Perkmann T, Winkler H, Graninger W, et al. Circulating levels of the interleukin (IL)-4 receptor and of IL-18 in patients with Plasmodium falciparum malaria. Cytokine 2005; 29 : 153–8.
  14. Nagamine Y, Hayano M, Kashiwamura S, et al. Involvement of interleukin-18 in severe Plasmodium falciparum malaria. Trans R Soc Trop Med Hyg 2003; 97 : 236–41.
  15. Hermsen CC, Konijnenberg Y, Mulder L, et al. Circulating concentrations of soluble granzyme A and B increase during natural and experimental Plasmodium falciparum infections. Clin Exp Immunol 2003; 132 : 467–72.
  16. Scragg IG, Hensmann M, Bate CA, et al. Early cytokine induction by Plasmodium falciparum is not a classical endotoxin-like process. Eur J Immunol 1999; 29 : 2636–44.
  17. Artavanis-Tsakonas K, Riley EM. Innate immune response to malaria: rapid induction of IFN-gamma from human NK cells by live Plasmodium falciparum-infected erythrocytes. J Immunol 2002; 169 : 2956–63.
  18. Artavanis-Tsakonas K, Eleme K, McQueen KL, et al. Activation of a subset of human NK cells upon contact with Plasmodium falciparum-infected erythrocytes. J Immunol 2003; 171 : 5396–405.
  19. Korbel DS, Newman KC, Almeida CR, et al. Heterogeneous human NK cell responses to Plasmodium falciparum-infected erythrocytes. J Immunol 2005; 175 : 7466–73.
  20. Baratin M, Roetynck S, Lepolard C, et al. Natural killer cell and macrophage cooperation in MyD88-dependent innate responses to Plasmodium falciparum. Proc Natl Acad Sci USA 2005; 102 : 14747–52.
  21. Ferlazzo G, Munz C. NK cell compartments and their activation by dendritic cells. J Immunol 2004; 172 : 1333–9.
  22. Stewart CA, Vivier E, Colonna M. Strategies of natural killer cell recognition and signaling. Curr Top Microbiol Immunol 2006; 298 : 1–21.
  23. Imai K, Matsuyama S, Miyake S, et al. Natural cytotoxic activity of peripheral-blood lymphocytes and cancer incidence: an 11-year follow-up study of a general population. Lancet 2000; 356 : 1795–9.
  24. Zitvogel L, Terme M, Borg C, et al. Dendritic cell-NK cell cross-talk: regulation and physiopathology. Curr Top Microbiol Immunol 2006; 298 : 157–74.
  25. Bottino C, Moretta L, Moretta A. NK cell activating receptors and tumor recognition in humans. Curr Top Microbiol Immunol 2006; 298 : 175–82.
  26. Orago AS, Facer CA. Cytotoxicity of human natural killer (NK) cell subsets for Plasmodium falciparum erythrocytic schizonts: stimulation by cytokines and inhibition by neomycin. Clin Exp Immunol 1991; 86 : 22–9.
  27. Mavoungou E, Luty AJ, Kremsner PG. Natural killer (NK) cell-mediated cytolysis of Plasmodium falciparum-infected human red blood cells in vitro. Eur Cytokine Netw 2003; 14 : 134–42.
  28. Rubio V, Stuge TB, Singh N, et al. Ex vivo identification, isolation and analysis of tumor-cytolytic T cells. Nat Med 2003; 9 : 1377–82.
  29. Alter G, Malenfant JM, Altfeld M. CD107a as a functional marker for the identification of natural killer cell activity. J Immunol Methods 2004; 294 : 15–22.
  30. Cooper MA, Fehniger TA, Caligiuri MA. The biology of human natural killer-cell subsets. Trends Immunol 2001; 22 : 633–40.
  31. Stevenson MM, Riley EM. Innate immunity to malaria. Nat Rev Immunol 2004; 4 : 169–80.
  32. Pichyangkul S, Yongvanitchit K, Kum-arb U, et al. Malaria blood stage parasites activate human plasmacytoid dendritic cells and murine dendritic cells through a Toll-like receptor 9-dependent pathway. J Immunol 2004; 172 : 4926–33.
  33. Urban BC, Ing R, Stevenson MM. Early interactions between blood-stage plasmodium parasites and the immune system. Curr Top Microbiol Immunol 2005; 297 : 25–70.
  34. Stevenson MM, Urban BC. Antigen presentation and dendritic cell biology in malaria. Parasite Immunol 2006; 28 : 5–14.
  35. Behr C, Poupot R, Peyrat MA, et al. Plasmodium falciparum stimuli for human gammadelta T cells are related to phosphorylated antigens of mycobacteria. Infect Immun 1996; 64 : 2892–6.
  36. Fournie JJ, Bonneville M. Stimulation of gamma delta T cells by phosphoantigens. Res Immunol 1996; 147 : 338–47.
  37. Farouk SE, Mincheva-Nilsson L, Krensky AM, et al. Gamma delta T cells inhibit in vitro growth of the asexual blood stages of Plasmodium falciparum by a granule exocytosis-dependent cytotoxic pathway that requires granulysin. Eur J Immunol 2004; 34 : 2248–56.
  38. Krishnegowda G, Hajjar AM, Zhu J, et al. Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: cell signaling receptors, glycosylphosphatidylinositol (GPI) structural requirement, and regulation of GPI activity. J Biol Chem 2005; 280 : 8606–16.
  39. Zhu J, Krishnegowda G, Gowda DC. Induction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: the requirement of extracellular signal-regulated kinase, p38, c-jun n-terminal kinase and nf-{kappa}b pathways for the expression of proinflammatory cytokines and nitric oxide. J Biol Chem 2005; 280 : 8617–27.
  40. Moretta A. The dialogue between human natural killer cells and dendritic cells. Curr Opin Immunol 2005; 17 : 306–11.

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