Ins(1,4,5)P3 : un messager pour entendre

Roberto Bruzzone; Martine Cohen-Salmon

doi:10.1051/medsci/2005216-7585

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Volume 21 / Numéro 6-7 (Juin–Juillet 2005)

Med Sci (Paris), 21 6-7 (2005) 585-588

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Numéro		Med Sci (Paris) Volume 21, Numéro 6-7, Juin–Juillet 2005


Page(s)		585 - 588
Section		Nouvelles
DOI		https://doi.org/10.1051/medsci/2005216-7585
Publié en ligne		15 juin 2005

Gerido DA, White TW. Connexin disorders of the ear, skin, and lens. Biochim Biophys Acta 2004; 1662 : 159–70. [Google Scholar]
Bruzzone R, White TW, Paul, DL. Connections with connexins : the molecular basis of direct intercellular signaling. Eur J Biochem 1996; 238 : 1–27. [Google Scholar]
Harris AL. Emerging issues of connexin channels : biophysics fills the gap. Q Rev Biophys 2001; 34 : 325–472. [Google Scholar]
Beltramello M, Piazza V, Bukauskas FF, et al. Impaired permeability to Ins(1,4,5)P3 in a mutant connexin underlies recessive hereditary deafness. Nat Cell Biol 2005; 7 : 63–9. [Google Scholar]
Petit C, Levilliers J, Hardelin JP. Molecular genetics of hearing loss. Annu Rev Genet 2001; 35 : 589–646. [Google Scholar]
Lautermann J, Ten Cate WJ, Altenhoff P, et al. Expression of the gap-junction connexins 26 and 30 in the rat cochlea. Cell Tissue Res 1998; 294 : 415–20. [Google Scholar]
Boettger T, Hubner CA, Maier H, et al. Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4. Nature 2002; 416 : 874–8. [Google Scholar]
Cohen-Salmon M, Ott T, Michel V, et al. Targeted ablation of connexin26 in the inner ear epithelial gap junction network causes hearing impairment and cell death. Curr Biol 2002; 12 : 1106–11. [Google Scholar]
Valiunas V, Manthey D, Vogel R, et al. Biophysical properties of mouse connexin30 gap junction channels studied in transfected human HeLa cells. J Physiol 1999; 519 : 631–44. [Google Scholar]
Bevans CG, Kordel M, Rhee SK, Harris AL. Isoform composition of connexin channels determines selectivity among second messengers and uncharged molecules. J Biol Chem 1998; 273 : 2808–16. [Google Scholar]
Goldberg GS, Lampe PD, Nicholson BJ. Selective transfer of endogenous metabolites through gap junctions composed of different connexins. Nat Cell Biol 1999; 1 : 457–9. [Google Scholar]
Niessen H, Harz H, Bedner P, et al. Selective permeability of different connexin channels to the second messenger inositol 1,4,5-trisphosphate. J Cell Sci 2000; 113 : 1365–72. [Google Scholar]
Bruzzone R, Gomès D, Veronesi V, et al. Functional analysis of recessive mutations of human connexin26 associated with nonsyndromic deafness. FEBS Lett 2003; 533 : 79–88. [Google Scholar]
Gale JE, Piazza V, Ciubotaru CD, Mammano F. A mechanism for sensing noise damage in the inner ear. Curr Biol 2004; 14 : 526–9. [Google Scholar]
Oh S, Ri Y, Bennett MV, et al. Changes in permeability caused by connexin 32 mutations underlie X-linked Charcot-Marie-Tooth disease. Neuron 1997; 19 : 927–38. [Google Scholar]

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[1] Gerido DA, White TW. Connexin disorders of the ear, skin, and lens. Biochim Biophys Acta 2004; 1662 : 159–70. [Google Scholar]

[2] Bruzzone R, White TW, Paul, DL. Connections with connexins : the molecular basis of direct intercellular signaling. Eur J Biochem 1996; 238 : 1–27. [Google Scholar]

[3] Harris AL. Emerging issues of connexin channels : biophysics fills the gap. Q Rev Biophys 2001; 34 : 325–472. [Google Scholar]

[4] Beltramello M, Piazza V, Bukauskas FF, et al. Impaired permeability to Ins(1,4,5)P3 in a mutant connexin underlies recessive hereditary deafness. Nat Cell Biol 2005; 7 : 63–9. [Google Scholar]

[5] Petit C, Levilliers J, Hardelin JP. Molecular genetics of hearing loss. Annu Rev Genet 2001; 35 : 589–646. [Google Scholar]

[6] Lautermann J, Ten Cate WJ, Altenhoff P, et al. Expression of the gap-junction connexins 26 and 30 in the rat cochlea. Cell Tissue Res 1998; 294 : 415–20. [Google Scholar]

[7] Boettger T, Hubner CA, Maier H, et al. Deafness and renal tubular acidosis in mice lacking the K-Cl co-transporter Kcc4. Nature 2002; 416 : 874–8. [Google Scholar]

[8] Cohen-Salmon M, Ott T, Michel V, et al. Targeted ablation of connexin26 in the inner ear epithelial gap junction network causes hearing impairment and cell death. Curr Biol 2002; 12 : 1106–11. [Google Scholar]

[9] Valiunas V, Manthey D, Vogel R, et al. Biophysical properties of mouse connexin30 gap junction channels studied in transfected human HeLa cells. J Physiol 1999; 519 : 631–44. [Google Scholar]

[10] Bevans CG, Kordel M, Rhee SK, Harris AL. Isoform composition of connexin channels determines selectivity among second messengers and uncharged molecules. J Biol Chem 1998; 273 : 2808–16. [Google Scholar]

[11] Goldberg GS, Lampe PD, Nicholson BJ. Selective transfer of endogenous metabolites through gap junctions composed of different connexins. Nat Cell Biol 1999; 1 : 457–9. [Google Scholar]

[12] Niessen H, Harz H, Bedner P, et al. Selective permeability of different connexin channels to the second messenger inositol 1,4,5-trisphosphate. J Cell Sci 2000; 113 : 1365–72. [Google Scholar]

[13] Bruzzone R, Gomès D, Veronesi V, et al. Functional analysis of recessive mutations of human connexin26 associated with nonsyndromic deafness. FEBS Lett 2003; 533 : 79–88. [Google Scholar]

[14] Gale JE, Piazza V, Ciubotaru CD, Mammano F. A mechanism for sensing noise damage in the inner ear. Curr Biol 2004; 14 : 526–9. [Google Scholar]

[15] Oh S, Ri Y, Bennett MV, et al. Changes in permeability caused by connexin 32 mutations underlie X-linked Charcot-Marie-Tooth disease. Neuron 1997; 19 : 927–38. [Google Scholar]