Accès gratuit
Numéro
Med Sci (Paris)
Volume 21, Numéro 1, Janvier 2005
Page(s) 55 - 60
Section M/S Revues
DOI https://doi.org/10.1051/medsci/200521155
Publié en ligne 15 janvier 2005
  1. Gordon RD, Klemm SA, Tunny TJ, et al. Gordon’s syndrome : a sodium-volume-dependent form of hypertension with a genetic basis. In : Laragh JH, Brenner BM, eds. Hypertension : pathophysiology, diagnosis, and management, 2nd ed. New York : Raven, Press Ltd, 1995 : 2111–3.
  2. Disse-Nicodeme S, Desitter I, Fiquet-Kempf B, et al. Genetic heterogeneity of familial hyperkalaemic hypertension. J Hypertens 2001; 19 : 1957–64.
  3. Wilson FH, Disse-Nicodeme S, Choate KA, et al. Human hypertension caused by mutations in WNK kinases. Science 2001; 293 : 1107–12.
  4. Xu B, English JM, Wilsbacher JL, et al. WNK1, a novel mammalian serine/threonine protein kinase lacking the catalytic lysine in subdomain II. J Biol Chem 2000; 275 : 16795–801.
  5. Xu BE, Min X, Stippec S, Lee BH, et al. Regulation of WNK1 by an autoinhibitory domain and autophosphorylation. J Biol Chem 2002; 277 : 48456–62.
  6. Verissimo F, Jordan P. WNK kinases, a novel protein kinase subfamily in multi-cellular organisms. Oncogene 2001; 20 : 5562–9.
  7. Wang Z, Yang CL, Ellison DH. Comparison of WNK4 and WNK1 kinase and inhibiting activities. Biochem Biophys Res Commun 2004; 317 : 939–44.
  8. Xu BE, Stippec S, Lenertz L, et al. WNK1 activates ERK5 by an MEKK2/3-dependent mechanism. J Biol Chem 2004; 279 : 7826–31.
  9. Delaloy C, Lu J, Houot AM, et al. Multiple promoters in the WNK1 gene : one controls expression of a kidney-specific kinase-defective isoform. Mol Cell Biol 2003; 23 : 9208–21.
  10. O’Reilly M, Marshall E, Speirs HJ, Brown RW. WNK1, a gene within a novel blood pressure control pathway, tissue-specifically generates radically different isoforms with and without a kinase domain. J Am Soc Nephrol 2003; 14 : 2447–56.
  11. Choate KA, Kahle KT, Wilson FH, et al. WNK1, a kinase mutated in inherited hypertension with hyperkalemia, localizes to diverse Cl-transporting epithelia. Proc Natl Acad Sci USA 2003; 100 : 663–8.
  12. Kahle KT, Gimenez I, Hassan H, et al. WNK4 regulates apical and basolateral Cl flux in extrarenal epithelia. Proc Natl Acad Sci USA 2004; 101 : 2064–9.
  13. Schnermann J. Sodium transport deficiency and sodium balance in gene-targeted mice. Acta Physiol Scand 2001; 173 : 59–66.
  14. Masilamani S, Wang X, Kim GH, et al. Time course of renal Na-K-ATPase, NHE3, NKCC2, NCC, and ENaC abundance changes with dietary NaCl restriction. Am J Physiol Renal Physiol 2002; 283 : 648–57.
  15. Wang W. Regulation of renal K transport by dietary K intake. Annu Rev Physiol 2004; 66 : 547–69.
  16. Yoo D, Kim BY, Campo C, et al. Cell surface expression of the ROMK (Kir 1.1) channel is regulated by the aldosterone-induced kinase, SGK-1, and protein kinase A. J Biol Chem 2003; 278 : 23066–75.
  17. Wilson FH, Kahle KT, Sabath E, et al. Molecular pathogenesis of inherited hypertension with hyperkalemia : the Na-Cl cotransporter is inhibited by wild-type but not mutant WNK4. Proc Natl Acad Sci USA 2003; 100 : 680–4.
  18. Yang CL, Angell J, Mitchell R, Ellison DH. WNK kinases regulate thiazide-sensitive Na-Cl cotransport. J Clin Invest 2003; 111 : 1039–45.
  19. Kahle KT, Wilson FH, Leng Q, et al. WNK4 regulates the balance between renal NaCl reabsorption and K+ secretion. Nat Genet 2003; 35 : 372–6.
  20. Schambelan M, Sebastian A, Rector FC Jr. Mineralocorticoid-resistant renal hyperkalemia without salt wasting (type II pseudohypoaldosteronism) : role of increased renal chloride reabsorption. Kidney Int 1981 : 19 : 716–27.
  21. Yamauchi K, Rai T, Kobayashi K, et al. Disease-causing mutant WNK4 increases paracellular chloride permeability and phosphorylates claudins. Proc Natl Acad Sci USA 2004; 101 : 4690–4.
  22. Kahle KT, MacGregor GG, Wilson FH, et al. Paracellular Cl– permeability is regulated by WNK4 kinase : Insight into normal physiology and hypertension. Proc Natl Acad Sci USA 2004; 101 : 14877–82.

Les statistiques affichées correspondent au cumul d'une part des vues des résumés de l'article et d'autre part des vues et téléchargements de l'article plein-texte (PDF, Full-HTML, ePub... selon les formats disponibles) sur la platefome Vision4Press.

Les statistiques sont disponibles avec un délai de 48 à 96 heures et sont mises à jour quotidiennement en semaine.

Le chargement des statistiques peut être long.