Des protéines télomériques participent à la persistance extra-chromosomique du génome du virus d’Epstein-Barr

Alain Sergeant; Louis Gazzolo

doi:10.1051/medsci/20021889812

Accès gratuit

Numéro		Med Sci (Paris) Volume 18, Numéro 8-9, Août–Septembre 2002


Page(s)		812 - 814
Section		Le Magazine : Nouvelles
DOI		https://doi.org/10.1051/medsci/20021889812
Publié en ligne		15 août 2002

Rickinson AB, Kieff E. Epstein-Barr virus. In : Fields BN, Knipe DM, Howley PM, eds. Virology. New York : Raven Press, 1996: 2397–446. [Google Scholar]
Babcock GJ, Decker MV, Thorley-Lawson DA. EBV persistence in memory B cells in vivo. Immunity 1998; 9 : 395–404. [Google Scholar]
Rickinson AB, Kieff E. Epstein-Barr virus. In : Fields BN, Knipe DM, Howley PM, eds. Virology. New York : Raven Press, 1996 : 2343–96. [Google Scholar]
Lupton S, Levine A. Mapping genetic elements of Epstein-Barr virus that facilitate extrachromosomal persistence of the Epstein-Barr virus-deri!ved plasmids in human cells. Mol Cell Biol 1985; 5 : 2533–42. [Google Scholar]
Yates JL, Camiolo SM, Bashaw JM. The minimal replicator of Epstein-Barr virus. J Virol 2000; 65 : 483–8. [Google Scholar]
Rawlins DR, Milman G, Hayward SD, Hayward GS. Sequence-specific DNA binding of the Epstein-Barr virus nuclear antigen 1 (EBNA1) to clustered sites in the plasmid maintenance region. Cell 1985; 42 : 859–68. [Google Scholar]
Deng Z, Lezina L, Chen C, Shtivelband S, So W, Lieberman PM. Telomeric proteins regulate episomal maintenance of Epstein-Barr virus origin of plasmid replication. Mol Cell 2002; 9 : 493–503. [Google Scholar]
Aiyar A, Tyree C, Sugden B. The plasmid replicon of EBV consists of multiple cis-acting elements that facilitate DNA synthesis by the cell and a viral maintenance element. EMBO J 1998; 17 : 6394–403. [Google Scholar]
Balckburn E. Switching and signalling at the telomere. Cell 2001; 106 : 661–73. [Google Scholar]
Longtime MS, Enomoto S, Finstad SL, Berman J. Yeast telomere repeat sequence (TRS) improves circular plasmid segregation, and TRS plasmid segregation involves the RAP1 gene product. Mol Cell Biol 1992; 12 : 1997–2009. [Google Scholar]

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[1] Rickinson AB, Kieff E. Epstein-Barr virus. In : Fields BN, Knipe DM, Howley PM, eds. Virology. New York : Raven Press, 1996: 2397–446. [Google Scholar]

[2] Babcock GJ, Decker MV, Thorley-Lawson DA. EBV persistence in memory B cells in vivo. Immunity 1998; 9 : 395–404. [Google Scholar]

[3] Rickinson AB, Kieff E. Epstein-Barr virus. In : Fields BN, Knipe DM, Howley PM, eds. Virology. New York : Raven Press, 1996 : 2343–96. [Google Scholar]

[4] Lupton S, Levine A. Mapping genetic elements of Epstein-Barr virus that facilitate extrachromosomal persistence of the Epstein-Barr virus-deri!ved plasmids in human cells. Mol Cell Biol 1985; 5 : 2533–42. [Google Scholar]

[5] Yates JL, Camiolo SM, Bashaw JM. The minimal replicator of Epstein-Barr virus. J Virol 2000; 65 : 483–8. [Google Scholar]

[6] Rawlins DR, Milman G, Hayward SD, Hayward GS. Sequence-specific DNA binding of the Epstein-Barr virus nuclear antigen 1 (EBNA1) to clustered sites in the plasmid maintenance region. Cell 1985; 42 : 859–68. [Google Scholar]

[7] Deng Z, Lezina L, Chen C, Shtivelband S, So W, Lieberman PM. Telomeric proteins regulate episomal maintenance of Epstein-Barr virus origin of plasmid replication. Mol Cell 2002; 9 : 493–503. [Google Scholar]

[8] Aiyar A, Tyree C, Sugden B. The plasmid replicon of EBV consists of multiple cis-acting elements that facilitate DNA synthesis by the cell and a viral maintenance element. EMBO J 1998; 17 : 6394–403. [Google Scholar]

[9] Balckburn E. Switching and signalling at the telomere. Cell 2001; 106 : 661–73. [Google Scholar]

[10] Longtime MS, Enomoto S, Finstad SL, Berman J. Yeast telomere repeat sequence (TRS) improves circular plasmid segregation, and TRS plasmid segregation involves the RAP1 gene product. Mol Cell Biol 1992; 12 : 1997–2009. [Google Scholar]